Stability of gene expression by primary bronchial epithelial cells over increasing passage number
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2466
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
Stability of gene expression by primary bronchial epithelial cells over increasing passage number ; volume:18 ; number:1 ; day:29 ; month:5 ; year:2018 ; pages:1-11 ; date:12.2018
BMC pulmonary medicine ; 18, Heft 1 (29.5.2018), 1-11, 12.2018
- Creator
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Reeves, Stephen R.
- Contributor
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Barrow, Kaitlyn A.
White, Maria P.
Rich, Lucille M.
Naushab, Maryam
Debley, Jason S.
SpringerLink (Online service)
- DOI
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10.1186/s12890-018-0652-2
- URN
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urn:nbn:de:101:1-2018080523375481205488
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:38 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Reeves, Stephen R.
- Barrow, Kaitlyn A.
- White, Maria P.
- Rich, Lucille M.
- Naushab, Maryam
- Debley, Jason S.
- SpringerLink (Online service)
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P38 MAPK and glucocorticoid receptor crosstalk in bronchial epithelial cells
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Xenobiotic metabolism in differentiated human bronchial epithelial cells
Bronchial epithelial gene expression and interstitial lung abnormalities
Altered microRNA expression profile during epithelial wound repair in bronchial epithelial cells
TWEAK enhances TGF-β-induced epithelial-mesenchymal transition in human bronchial epithelial cells
circ_0038467 promotes PM2.5-induced bronchial epithelial cell dysfunction
Mechanical compression attenuates normal human bronchial epithelial wound healing
Quercitrin protects human bronchial epithelial cells from oxidative damage
β2 adrenergic agonist suppresses eosinophil-induced epithelial-to-mesenchymal transition of bronchial epithelial cells
Endoplasmic reticulum stress increases inflammatory responses by bronchial epithelial cells
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