Amelioration of experimental autoimmune encephalomyelitis by clozapine is not associated with defective CD4 T cell responses
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1742-2094
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
Amelioration of experimental autoimmune encephalomyelitis by clozapine is not associated with defective CD4 T cell responses ; volume:14 ; number:1 ; day:29 ; month:3 ; year:2017 ; pages:1-10 ; date:12.2017
Journal of neuroinflammation ; 14, Heft 1 (29.3.2017), 1-10, 12.2017
- Keyword
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Immunologie
Immunsystem
Lymphozyt
Leukozyt
Immunität
Resistenz
Immunchemie
- Creator
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Zareie, Pirooz
- Contributor
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Connor, Bronwen
La Flamme, Anne Camille
SpringerLink (Online service)
- DOI
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10.1186/s12974-017-0842-5
- URN
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urn:nbn:de:1111-2017041613939
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:34 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Zareie, Pirooz
- Connor, Bronwen
- La Flamme, Anne Camille
- SpringerLink (Online service)
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Clozapine reduces infiltration into the CNS by targeting migration in experimental autoimmune encephalomyelitis
Amelioration of experimental autoimmune encephalomyelitis by in vivo reprogramming of macrophages using pro-resolving factors
Clozapine-induced agranulocytosis
Successful clozapine rechallenge following recurrent clozapine-associated pancreatitis: a case report
Clinical and Histopathological Amelioration of Experimental Autoimmune Encephalomyelitis by AAV Vectors Expressing a Soluble Interleukin-23 Receptor
Pathogenic function of bystander-activated memory-like CD4+ T cells in autoimmune encephalomyelitis
Microglia-regulated activation of CD4+ T cells in the development of experimental autoimmune encephalomyelitis
Defective classes
Defective response of CD4+T cells to retinoic acid and TGFβ in systemic lupus erythematosus
The DREADD agonist clozapine N-oxide (CNO) is reverse-metabolized to clozapine and produces clozapine-like interoceptive stimulus effects in rats and mice
The lived experience of clozapine discontinuation in patients and carers following suspected clozapine-induced neutropenia
Clozapine regulates microglia and is effective in chronic experimental autoimmune encephalomyelitis
Clozapine reduces infiltration into the CNS by targeting migration in experimental autoimmune encephalomyelitis
Amelioration of experimental autoimmune encephalomyelitis by in vivo reprogramming of macrophages using pro-resolving factors
Clozapine-induced agranulocytosis
Successful clozapine rechallenge following recurrent clozapine-associated pancreatitis: a case report
Clinical and Histopathological Amelioration of Experimental Autoimmune Encephalomyelitis by AAV Vectors Expressing a Soluble Interleukin-23 Receptor
Pathogenic function of bystander-activated memory-like CD4+ T cells in autoimmune encephalomyelitis
Microglia-regulated activation of CD4+ T cells in the development of experimental autoimmune encephalomyelitis
Defective classes
Defective response of CD4+T cells to retinoic acid and TGFβ in systemic lupus erythematosus
The DREADD agonist clozapine N-oxide (CNO) is reverse-metabolized to clozapine and produces clozapine-like interoceptive stimulus effects in rats and mice