PARP1 and XRCC1 exhibit a reciprocal relationship in genotoxic stress response
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Erschienen in
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In: Cell Biology and Toxicology. Springer. 2023, 39, pp. 345-364. ISSN 0742-2091. eISSN 1573-6822. Available under: doi: 10.1007/s10565-022-09739-9
- Klassifikation
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Biowissenschaften, Biologie
- Ereignis
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Veröffentlichung
- (wo)
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Konstanz
- (wer)
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KOPS Universität Konstanz
- (wann)
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2023
- Urheber
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Reber, Julia M.
Božić-Petković, Jovana
Lippmann, Michelle
Mazzardo, Marvin
Dilger, Asisa
Warmers, Rebecca
Bürkle, Alexander
Mangerich, Aswin
- URN
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urn:nbn:de:bsz:352-2-17a6m9s572cvg9
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:47 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
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Beteiligte
- Reber, Julia M.
- Božić-Petković, Jovana
- Lippmann, Michelle
- Mazzardo, Marvin
- Dilger, Asisa
- Warmers, Rebecca
- Bürkle, Alexander
- Mangerich, Aswin
- KOPS Universität Konstanz
Entstanden
- 2023
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Untersuchungen zur Induktion von zellulärem DNA-Repair durch Herpes-simplex-Virus-Infektion
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Real-time monitoring of PARP1-dependent PARylation by ATR-FTIR spectroscopy
Poly(ADP-ribose)-mediated interplay of XPA and PARP1 leads to reciprocal regulation of protein function
Epigenetic and redox biomarkers : Novel insights from the MARK-AGE study
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How to kill tumor cells with inhibitors of poly(ADP-ribosyl)ation
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PARP1 protects from benzo[a]pyrene diol epoxide-induced replication stress and mutagenicity
PARP1 and XRCC1 exhibit a reciprocal relationship in genotoxic stress response
Untersuchungen zur Induktion von zellulärem DNA-Repair durch Herpes-simplex-Virus-Infektion
Pleiotropic Cellular Functions of PARP1 in Longevity and Aging : Genome Maintenance Meets Inflammation
Real-time monitoring of PARP1-dependent PARylation by ATR-FTIR spectroscopy
Poly(ADP-ribose)-mediated interplay of XPA and PARP1 leads to reciprocal regulation of protein function
Epigenetic and redox biomarkers : Novel insights from the MARK-AGE study
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How to kill tumor cells with inhibitors of poly(ADP-ribosyl)ation
Poly(ADP-ribose) : PARadigms and PARadoxes
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