A CCD-based reader combined with CdS quantum dot-labeled lateral flow strips for ultrasensitive quantitative detection of CagA
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1556-276X
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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A CCD-based reader combined with CdS quantum dot-labeled lateral flow strips for ultrasensitive quantitative detection of CagA ; volume:9 ; number:1 ; day:4 ; month:2 ; year:2014 ; pages:1-8 ; date:12.2014
Nanoscale research letters ; 9, Heft 1 (4.2.2014), 1-8, 12.2014
- Creator
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Gui, Chen
Wang, Kan
Li, Chao
Dai, Xuan
Cui, Daxiang
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/1556-276X-9-57
- URN
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urn:nbn:de:101:1-2021081408331323161314
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 11:03 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Gui, Chen
- Wang, Kan
- Li, Chao
- Dai, Xuan
- Cui, Daxiang
- SpringerLink (Online service)
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Integrin-independent Helicobacter pylori CagA translocation via type IV secretion
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VacA promotes CagA accumulation in gastric epithelial cells during Helicobacter pylori infection
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Interaktion des Cag-TypIV-Sekretionssystems von Helicobacter pylori mit β1-Integrin zur Untersuchung des Translokationsmechanismus des Cytotoxin-assoziierten Antigens (CagA)
Functional antagonism between CagA and DLC1 in gastric cancer
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Die Rolle der Tyrosinphosphorylierungsmotive des Onkoproteins CagA von Helicobacter pylori
Die Rolle der Tyrosinphosphorylierungsmotive des Onkoproteins CagA von Helicobacter pylori
Integrin-independent Helicobacter pylori CagA translocation via type IV secretion
Impact of Helicobacter pylori CagA on (canonical) Wnt/β-catenin Signaling
Helicobacter pylori cagA and vacA genes in dyspeptic Ghanaian patients
Helicobacter pylori CagL Y58/E59 Mutation Turns-Off Type IV Secretion-Dependent Delivery of CagA into Host Cells
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