Accumulation of potential driver genes with genomic alterations predicts survival of high-risk neuroblastoma patients
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1745-6150
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Accumulation of potential driver genes with genomic alterations predicts survival of high-risk neuroblastoma patients ; volume:13 ; number:1 ; day:16 ; month:7 ; year:2018 ; pages:1-11 ; date:1.2018
Biology direct ; 13, Heft 1 (16.7.2018), 1-11, 1.2018
- Creator
-
Suo, Chen
- Contributor
-
Deng, Wenjiang
Vu, Trung Nghia
Li, Mingrui
Shi, Leming
Pawitan, Yudi
SpringerLink (Online service)
- DOI
-
10.1186/s13062-018-0218-5
- URN
-
urn:nbn:de:101:1-2018092122280727773525
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:24 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Suo, Chen
- Deng, Wenjiang
- Vu, Trung Nghia
- Li, Mingrui
- Shi, Leming
- Pawitan, Yudi
- SpringerLink (Online service)
Other Objects (12)
Allele-specific expression reveals genes with recurrent cis-regulatory alterations in high-risk neuroblastoma
Acquired genetic alterations in tumor cells dictate the development of high-risk neuroblastoma and clinical outcomes
Genetic alterations and resectability predict outcome in patients with neuroblastoma assigned to high-risk solely by MYCN amplification
Macrophage-mediated anti-tumor immunity against high-risk neuroblastoma
Artificial neural network classifier predicts neuroblastoma patients’ outcome
Novel therapeutic strategies targeting telomere maintenance mechanisms in high-risk neuroblastoma
Pre-clinical investigation of astatine-211-parthanatine for high-risk neuroblastoma
Epigenetic alterations differ in phenotypically distinct human neuroblastoma cell lines
SNHG25 facilitates SNORA50C accumulation to stabilize HDAC1 in neuroblastoma cells
Functional validation of novel target structures for treatment of high-risk neuroblastoma
Transcribed-ultra conserved region expression is associated with outcome in high-risk neuroblastoma
Identification and characterization of genetic alterations in neuroblastoma and their prognostic relevance
Allele-specific expression reveals genes with recurrent cis-regulatory alterations in high-risk neuroblastoma
Acquired genetic alterations in tumor cells dictate the development of high-risk neuroblastoma and clinical outcomes
Genetic alterations and resectability predict outcome in patients with neuroblastoma assigned to high-risk solely by MYCN amplification
Macrophage-mediated anti-tumor immunity against high-risk neuroblastoma
Artificial neural network classifier predicts neuroblastoma patients’ outcome
Novel therapeutic strategies targeting telomere maintenance mechanisms in high-risk neuroblastoma
Pre-clinical investigation of astatine-211-parthanatine for high-risk neuroblastoma
Epigenetic alterations differ in phenotypically distinct human neuroblastoma cell lines
SNHG25 facilitates SNORA50C accumulation to stabilize HDAC1 in neuroblastoma cells
Functional validation of novel target structures for treatment of high-risk neuroblastoma
Transcribed-ultra conserved region expression is associated with outcome in high-risk neuroblastoma
Identification and characterization of genetic alterations in neuroblastoma and their prognostic relevance
Allele-specific expression reveals genes with recurrent cis-regulatory alterations in high-risk neuroblastoma
Acquired genetic alterations in tumor cells dictate the development of high-risk neuroblastoma and clinical outcomes
Genetic alterations and resectability predict outcome in patients with neuroblastoma assigned to high-risk solely by MYCN amplification
Macrophage-mediated anti-tumor immunity against high-risk neuroblastoma
Artificial neural network classifier predicts neuroblastoma patients’ outcome
Novel therapeutic strategies targeting telomere maintenance mechanisms in high-risk neuroblastoma
Pre-clinical investigation of astatine-211-parthanatine for high-risk neuroblastoma
Epigenetic alterations differ in phenotypically distinct human neuroblastoma cell lines
SNHG25 facilitates SNORA50C accumulation to stabilize HDAC1 in neuroblastoma cells
Functional validation of novel target structures for treatment of high-risk neuroblastoma
Transcribed-ultra conserved region expression is associated with outcome in high-risk neuroblastoma