Spatiotemporal dynamics of homologous recombination repair at single collapsed replication forks
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2041-1723
- Umfang
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Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Spatiotemporal dynamics of homologous recombination repair at single collapsed replication forks ; volume:9 ; number:1 ; day:24 ; month:9 ; year:2018 ; pages:1-15 ; date:12.2018
Nature Communications ; 9, Heft 1 (24.9.2018), 1-15, 12.2018
- Klassifikation
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Biowissenschaften, Biologie
- Urheber
-
Whelan, Donna R.
- Beteiligte Personen und Organisationen
-
Lee, Wei Ting C.
Yin, Yandong
Ofri, Dylan M.
Bermudez-Hernandez, Keria
Keegan, Sarah
Fenyo, David
Rothenberg, Eli
SpringerLink (Online service)
- DOI
-
10.1038/s41467-018-06435-3
- URN
-
urn:nbn:de:101:1-2018110415575452357250
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:50 MESZ
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Beteiligte
- Whelan, Donna R.
- Lee, Wei Ting C.
- Yin, Yandong
- Ofri, Dylan M.
- Bermudez-Hernandez, Keria
- Keegan, Sarah
- Fenyo, David
- Rothenberg, Eli
- SpringerLink (Online service)
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Structural basis of homologous recombination
Dianhydrogalactitol induces replication-dependent DNA damage in tumor cells preferentially resolved by homologous recombination
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ATM orchestrates the DNA-damage response to counter toxic non-homologous end-joining at broken replication forks
Leveraging homologous recombination repair deficiency in sarcoma
Targeting homologous recombination deficiency in uterine leiomyosarcoma
Opposing roles for 53BP1 during homologous recombination
Homologous recombination defects and how they affect replication fork maintenance
Prevention of unwanted recombination at damaged replication forks
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DSS1 restrains BRCA2’s engagement with dsDNA for homologous recombination, replication fork protection, and R-loop homeostasis
Replication termination without a replication fork trap
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Dianhydrogalactitol induces replication-dependent DNA damage in tumor cells preferentially resolved by homologous recombination
Homologous recombination deficiency in breast cancer
ATM orchestrates the DNA-damage response to counter toxic non-homologous end-joining at broken replication forks
Leveraging homologous recombination repair deficiency in sarcoma
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