Characterisation of genetic regulatory effects for osteoporosis risk variants in human osteoclasts

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1474-760X

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Characterisation of genetic regulatory effects for osteoporosis risk variants in human osteoclasts ; volume:21 ; number:1 ; day:26 ; month:3 ; year:2020 ; pages:1-13 ; date:12.2020
Genome biology ; 21, Heft 1 (26.3.2020), 1-13, 12.2020

Creator: Mullin, Benjamin H.
Tickner, Jennifer
Zhu, Kun
Kenny, Jacob
Mullin, Shelby
Brown, Suzanne J.
Dudbridge, Frank
Pavlos, Nathan J.
Mocarski, Edward S.
Walsh, John P.
Xu, Jiake
Wilson, Scott G.

Contributor: SpringerLink (Online service)

DOI: 10.1186/s13059-020-01997-2

URN: urn:nbn:de:101:1-2020061623460518531444

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:45 AM CEST

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Associated

Mullin, Benjamin H.
Tickner, Jennifer
Zhu, Kun
Kenny, Jacob
Mullin, Shelby
Brown, Suzanne J.
Dudbridge, Frank
Pavlos, Nathan J.
Mocarski, Edward S.
Walsh, John P.
Xu, Jiake
Wilson, Scott G.
SpringerLink (Online service)

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Characterisation of genetic regulatory effects for osteoporosis risk variants in human osteoclasts

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Other Objects (12)

Smart osteoclasts targeted nanomedicine based on amorphous CaCO3 for effective osteoporosis reversal

Metabolic reprogramming of osteoclasts represents a therapeutic target during the treatment of osteoporosis

Metabolic reprogramming of osteoclasts represents a therapeutic target during the treatment of osteoporosis

LncRNA TUG1 was upregulated in osteoporosis and regulates the proliferation and apoptosis of osteoclasts

Retraction Note: LncRNA TUG1 was upregulated in osteoporosis and regulates the proliferation and apoptosis of osteoclasts

Comparative transcriptomic analysis identifies distinct molecular signatures and regulatory networks of chondroclasts and osteoclasts

Interpreting regulatory variants with predictive models

Giant osteoclasts in patients under bisphosphonates

Cell-cell fusion: human multinucleated osteoclasts

Angelicin improves osteoporosis in ovariectomized rats by reducing ROS production in osteoclasts through regulation of the KAT6A/Nrf2 signalling pathway

Negative feedback buffers effects of regulatory variants

Down-regulation of FTX promotes the differentiation of osteoclasts in osteoporosis through the Notch1 signaling pathway by targeting miR-137

Smart osteoclasts targeted nanomedicine based on amorphous CaCO3 for effective osteoporosis reversal

Metabolic reprogramming of osteoclasts represents a therapeutic target during the treatment of osteoporosis

Metabolic reprogramming of osteoclasts represents a therapeutic target during the treatment of osteoporosis

LncRNA TUG1 was upregulated in osteoporosis and regulates the proliferation and apoptosis of osteoclasts

Retraction Note: LncRNA TUG1 was upregulated in osteoporosis and regulates the proliferation and apoptosis of osteoclasts

Comparative transcriptomic analysis identifies distinct molecular signatures and regulatory networks of chondroclasts and osteoclasts

Interpreting regulatory variants with predictive models

Giant osteoclasts in patients under bisphosphonates

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Angelicin improves osteoporosis in ovariectomized rats by reducing ROS production in osteoclasts through regulation of the KAT6A/Nrf2 signalling pathway

Negative feedback buffers effects of regulatory variants

Down-regulation of FTX promotes the differentiation of osteoclasts in osteoporosis through the Notch1 signaling pathway by targeting miR-137

Smart osteoclasts targeted nanomedicine based on amorphous CaCO3 for effective osteoporosis reversal

Metabolic reprogramming of osteoclasts represents a therapeutic target during the treatment of osteoporosis

Metabolic reprogramming of osteoclasts represents a therapeutic target during the treatment of osteoporosis

LncRNA TUG1 was upregulated in osteoporosis and regulates the proliferation and apoptosis of osteoclasts

Retraction Note: LncRNA TUG1 was upregulated in osteoporosis and regulates the proliferation and apoptosis of osteoclasts

Comparative transcriptomic analysis identifies distinct molecular signatures and regulatory networks of chondroclasts and osteoclasts

Interpreting regulatory variants with predictive models

Giant osteoclasts in patients under bisphosphonates

Cell-cell fusion: human multinucleated osteoclasts

Angelicin improves osteoporosis in ovariectomized rats by reducing ROS production in osteoclasts through regulation of the KAT6A/Nrf2 signalling pathway

Negative feedback buffers effects of regulatory variants

Down-regulation of FTX promotes the differentiation of osteoclasts in osteoporosis through the Notch1 signaling pathway by targeting miR-137

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