Dominant optic atrophy, OPA1, and mitochondrial quality control: understanding mitochondrial network dynamics

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1750-1326

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Dominant optic atrophy, OPA1, and mitochondrial quality control: understanding mitochondrial network dynamics ; volume:8 ; number:1 ; day:25 ; month:9 ; year:2013 ; pages:1-12 ; date:12.2013
Molecular neurodegeneration ; 8, Heft 1 (25.9.2013), 1-12, 12.2013

Creator: Alavi, Marcel V.

Contributor: Fuhrmann, Nico
SpringerLink (Online service)

DOI: 10.1186/1750-1326-8-32

URN: urn:nbn:de:1111-2016073072140

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 02.08.2025, 11:00 AM CEST

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Alavi, Marcel V.
Fuhrmann, Nico
SpringerLink (Online service)

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Dominant optic atrophy, OPA1, and mitochondrial quality control: understanding mitochondrial network dynamics

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Other Objects (12)

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OPA1-related dominant optic atrophy is not strongly influenced by mitochondrial DNA background

The cristae modulator Optic atrophy 1 requires mitochondrial ATP synthase oligomers to safeguard mitochondrial function

Mitochondrial fusion mediator optic atrophy 1 as regulator of cell metabolism in macrophages

Mitochondrial dysfunction: roles in skeletal muscle atrophy

Characteristics of autosomal dominant WFS1-associated optic neuropathy and its comparability to OPA1-associated autosomal dominant optic atrophy

Resistance of mitochondrial p53 to dominant inhibition

Dominant mutations in MIEF1 affect mitochondrial dynamics and cause a singular late onset optic neuropathy

A neurodegenerative perspective on mitochondrial optic neuropathies

Mitochondrial pathophysiology beyond the retinal ganglion cell: occipital GABA is decreased in autosomal dominant optic neuropathy

Genomic deletions in OPA1 in Danish patients with autosomal dominant optic atrophy

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Dominant optic atrophy

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The cristae modulator Optic atrophy 1 requires mitochondrial ATP synthase oligomers to safeguard mitochondrial function

Mitochondrial fusion mediator optic atrophy 1 as regulator of cell metabolism in macrophages

Mitochondrial dysfunction: roles in skeletal muscle atrophy

Characteristics of autosomal dominant WFS1-associated optic neuropathy and its comparability to OPA1-associated autosomal dominant optic atrophy

Resistance of mitochondrial p53 to dominant inhibition

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The cristae modulator Optic atrophy 1 requires mitochondrial ATP synthase oligomers to safeguard mitochondrial function

Mitochondrial fusion mediator optic atrophy 1 as regulator of cell metabolism in macrophages

Mitochondrial dysfunction: roles in skeletal muscle atrophy

Characteristics of autosomal dominant WFS1-associated optic neuropathy and its comparability to OPA1-associated autosomal dominant optic atrophy

Resistance of mitochondrial p53 to dominant inhibition

Dominant mutations in MIEF1 affect mitochondrial dynamics and cause a singular late onset optic neuropathy

A neurodegenerative perspective on mitochondrial optic neuropathies

Mitochondrial pathophysiology beyond the retinal ganglion cell: occipital GABA is decreased in autosomal dominant optic neuropathy

Genomic deletions in OPA1 in Danish patients with autosomal dominant optic atrophy

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