B-Zellimmunität bei Primären Immundefekten wie den Class Switch Recombination (CSR)-Defekten und dem STAT3-Hyper-IgE-Syndrom
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Deutsch
- Notes
-
München, Ludwig-Maximilians-Universität, Dissertation, 2022
- Keyword
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Corporate Social Responsibility
Primärer Immundefekt
- Event
-
Veröffentlichung
- (where)
-
München
- (who)
-
Universitätsbibliothek der Ludwig-Maximilians-Universität
- (when)
-
2022
- Creator
- Contributor
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Renner, Ellen
- DOI
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10.5282/edoc.29653
- URN
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urn:nbn:de:bvb:19-296532
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:22 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Krätz, Carolin
- Renner, Ellen
- Universitätsbibliothek der Ludwig-Maximilians-Universität
Time of origin
- 2022
Other Objects (12)
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Identification and characterization of novel class switch recombination factors
STAT3 Hyper-IgE Syndrome—an Update and Unanswered Questions
SHLD1 is dispensable for 53BP1-dependent V(D)J recombination but critical for productive class switch recombination
Dissecting the composition, dynamics, and regulation of immunoglobulin class switch recombination
Predicting class switch recombination in B‐cells from antibody repertoire data
Molecular Assessment of Staphylococcus Aureus Strains in STAT3 Hyper-IgE Syndrome Patients
Molecular assessment of staphylococcus aureus strains in STAT3 hyper-IgE syndrome patients
Proteins mediating somatic hypermutation and class switch recombination at the immunoglobulin locus
Interventional pulmonary procedures and their outcomes in patients with STAT3 hyper IgE syndrome
Hyper-recombination in ribosomal DNA is driven by long-range resection-independent RAD51 accumulation
STAT3 – the switch of melanoma-associated NRAS mutations
Gene Editing in the STAT3 Hyper-IgE Syndrome
Identification and characterization of novel class switch recombination factors
STAT3 Hyper-IgE Syndrome—an Update and Unanswered Questions
SHLD1 is dispensable for 53BP1-dependent V(D)J recombination but critical for productive class switch recombination
Dissecting the composition, dynamics, and regulation of immunoglobulin class switch recombination
Predicting class switch recombination in B‐cells from antibody repertoire data
Molecular Assessment of Staphylococcus Aureus Strains in STAT3 Hyper-IgE Syndrome Patients
Molecular assessment of staphylococcus aureus strains in STAT3 hyper-IgE syndrome patients
Proteins mediating somatic hypermutation and class switch recombination at the immunoglobulin locus
Interventional pulmonary procedures and their outcomes in patients with STAT3 hyper IgE syndrome
Hyper-recombination in ribosomal DNA is driven by long-range resection-independent RAD51 accumulation
STAT3 – the switch of melanoma-associated NRAS mutations
Gene Editing in the STAT3 Hyper-IgE Syndrome
Identification and characterization of novel class switch recombination factors
STAT3 Hyper-IgE Syndrome—an Update and Unanswered Questions
SHLD1 is dispensable for 53BP1-dependent V(D)J recombination but critical for productive class switch recombination
Dissecting the composition, dynamics, and regulation of immunoglobulin class switch recombination
Predicting class switch recombination in B‐cells from antibody repertoire data
Molecular Assessment of Staphylococcus Aureus Strains in STAT3 Hyper-IgE Syndrome Patients
Molecular assessment of staphylococcus aureus strains in STAT3 hyper-IgE syndrome patients
Proteins mediating somatic hypermutation and class switch recombination at the immunoglobulin locus
Interventional pulmonary procedures and their outcomes in patients with STAT3 hyper IgE syndrome