- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1478-6354
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
C/EBP homologous protein drives pro-catabolic responses in chondrocytes ; volume:15 ; number:6 ; day:19 ; month:12 ; year:2013 ; pages:1-10 ; date:12.2013
Arthritis Research & Therapy ; 15, Heft 6 (19.12.2013), 1-10, 12.2013
- Creator
-
Husa, Matt
- Contributor
-
Petursson, Freyr
Lotz, Martin
Terkeltaub, Robert
Liu-Bryan, Ru
SpringerLink (Online service)
- DOI
-
10.1186/ar4415
- URN
-
urn:nbn:de:1111-2016051825189
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:51 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Husa, Matt
- Petursson, Freyr
- Lotz, Martin
- Terkeltaub, Robert
- Liu-Bryan, Ru
- SpringerLink (Online service)
Other Objects (12)
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Effects of hypoxia on anabolic and catabolic gene expression and DNA methylation in OA chondrocytes
Catabolic and proinflammatory effects of leptin in chondrocytes are regulated by suppressor of cytokine signaling-3
Secretome analysis of chondroitin sulfate-treated chondrocytes reveals anti-angiogenic, anti-inflammatory and anti-catabolic properties
Catabolic mediators from TLR2-mediated proteoglycan aggrecan peptide-stimulated chondrocytes are reduced by Lactobacillus-conditioned media
Upregulated endonuclease Regnase-1 suppresses osteoarthritis by forming a negative feedback loop of catabolic signaling in chondrocytes
MicroRNA-365 regulates IL-1β-induced catabolic factor expression by targeting HIF-2α in primary chondrocytes
Acute catabolic state
C/EBP homologous protein (CHOP) as a modifier of alpha-1 antitrypsin deficiency related liver disease development
Norepinephrine modulates IL-1β-induced catabolic response of human chondrocytes
Anabolic and catabolic responses of human articular chondrocytes to varying oxygen percentages
Silymarin modulates catabolic cytokine expression through Sirt1 and SOX9 in human articular chondrocytes
EBP TEAM
Effects of hypoxia on anabolic and catabolic gene expression and DNA methylation in OA chondrocytes
Catabolic and proinflammatory effects of leptin in chondrocytes are regulated by suppressor of cytokine signaling-3
Secretome analysis of chondroitin sulfate-treated chondrocytes reveals anti-angiogenic, anti-inflammatory and anti-catabolic properties
Catabolic mediators from TLR2-mediated proteoglycan aggrecan peptide-stimulated chondrocytes are reduced by Lactobacillus-conditioned media
Upregulated endonuclease Regnase-1 suppresses osteoarthritis by forming a negative feedback loop of catabolic signaling in chondrocytes
MicroRNA-365 regulates IL-1β-induced catabolic factor expression by targeting HIF-2α in primary chondrocytes
Acute catabolic state
C/EBP homologous protein (CHOP) as a modifier of alpha-1 antitrypsin deficiency related liver disease development
Norepinephrine modulates IL-1β-induced catabolic response of human chondrocytes
Anabolic and catabolic responses of human articular chondrocytes to varying oxygen percentages
Silymarin modulates catabolic cytokine expression through Sirt1 and SOX9 in human articular chondrocytes
EBP TEAM
Effects of hypoxia on anabolic and catabolic gene expression and DNA methylation in OA chondrocytes
Catabolic and proinflammatory effects of leptin in chondrocytes are regulated by suppressor of cytokine signaling-3
Secretome analysis of chondroitin sulfate-treated chondrocytes reveals anti-angiogenic, anti-inflammatory and anti-catabolic properties
Catabolic mediators from TLR2-mediated proteoglycan aggrecan peptide-stimulated chondrocytes are reduced by Lactobacillus-conditioned media
Upregulated endonuclease Regnase-1 suppresses osteoarthritis by forming a negative feedback loop of catabolic signaling in chondrocytes
MicroRNA-365 regulates IL-1β-induced catabolic factor expression by targeting HIF-2α in primary chondrocytes
Acute catabolic state