KAchannels reduce dendritic depolarization from synchronized synaptic input: implication for neural processing and epilepsy
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2202
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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KAchannels reduce dendritic depolarization from synchronized synaptic input: implication for neural processing and epilepsy ; volume:9 ; number:1 ; day:11 ; month:7 ; year:2008 ; pages:1-2 ; date:7.2008
BMC neuroscience ; 9, Heft 1 (11.7.2008), 1-2, 7.2008
- Creator
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Tigerholm, Jenny
- Contributor
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Fransén, Erik
SpringerLink (Online service)
- DOI
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10.1186/1471-2202-9-S1-P45
- URN
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urn:nbn:de:1111-2016032058470
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:54 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Tigerholm, Jenny
- Fransén, Erik
- SpringerLink (Online service)
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TRPM4-dependent post-synaptic depolarization is essential for the induction of NMDA receptor-dependent LTP in CA1 hippocampal neurons
Ion-concentration gradients induced by synaptic input increase the voltage depolarization in dendritic spines
The altered expression of cytoskeletal and synaptic remodeling proteins during epilepsy
Sustained Depolarization Induces Gene Expression Pattern Changes Related to Synaptic Plasticity in a Human Cholinergic Cellular Model
Vasospasm-Induced Spreading Depolarization and/or Spreading-Depolarization-Induced Vasospasm After Subarachnoid Hemorrhage
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