URMC-099 facilitates amyloid-β clearance in a murine model of Alzheimer’s disease

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1742-2094

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: URMC-099 facilitates amyloid-β clearance in a murine model of Alzheimer’s disease ; volume:15 ; number:1 ; day:5 ; month:5 ; year:2018 ; pages:1-14 ; date:12.2018
Journal of neuroinflammation ; 15, Heft 1 (5.5.2018), 1-14, 12.2018

Creator: Kiyota, Tomomi

Contributor: Machhi, Jatin
Lu, Yaman
Dyavarshetty, Bhagyalaxmi
Nemati, Maryam
Zhang, Gang
Mosley, R. Lee
Gelbard, Harris A.
Gendelman, Howard E.
SpringerLink (Online service)

DOI: 10.1186/s12974-018-1172-y

URN: urn:nbn:de:101:1-2018081323344246857699

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:21 AM CEST

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Kiyota, Tomomi
Machhi, Jatin
Lu, Yaman
Dyavarshetty, Bhagyalaxmi
Nemati, Maryam
Zhang, Gang
Mosley, R. Lee
Gelbard, Harris A.
Gendelman, Howard E.
SpringerLink (Online service)

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URMC-099 facilitates amyloid-β clearance in a murine model of Alzheimer’s disease

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Other Objects (12)

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Nanoscale structure of amyloid-β plaques in Alzheimer’s disease

The mixed-lineage kinase 3 inhibitor URMC-099 facilitates microglial amyloid-β degradation

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Medin co-aggregates with vascular amyloid-β in Alzheimer’s disease

Naringenin ameliorates amyloid-β pathology and neuroinflammation in Alzheimer’s disease

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Heparanase overexpression impedes perivascular clearance of amyloid-β from murine brain: relevance to Alzheimer’s disease

Nanoscale structure of amyloid-β plaques in Alzheimer’s disease

The mixed-lineage kinase 3 inhibitor URMC-099 facilitates microglial amyloid-β degradation

Rejuvenating aged microglia by p16ink4a-siRNA-loaded nanoparticles increases amyloid-β clearance in animal models of Alzheimer’s disease

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Naringenin ameliorates amyloid-β pathology and neuroinflammation in Alzheimer’s disease

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Distinct amyloid-β and tau-associated microglia profiles in Alzheimer’s disease

Myelin dysfunction drives amyloid-β deposition in models of Alzheimer’s disease

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Nanoscale structure of amyloid-β plaques in Alzheimer’s disease

The mixed-lineage kinase 3 inhibitor URMC-099 facilitates microglial amyloid-β degradation

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Naringenin ameliorates amyloid-β pathology and neuroinflammation in Alzheimer’s disease

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