Physical activity attenuates postprandial hyperglycaemia in homozygous TBC1D4 loss-of-function mutation carriers

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1432-0428

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Physical activity attenuates postprandial hyperglycaemia in homozygous TBC1D4 loss-of-function mutation carriers ; day:29 ; month:4 ; year:2021 ; pages:1-10
Diabetologia ; (29.4.2021), 1-10

Creator: Schnurr, Theresia M.
Jørsboe, Emil
Chadt, Alexandra
Dahl-Petersen, Inger K.
Kristensen, Jonas M.
Wojtaszewski, Jørgen F. P.
Springer, Christian
Bjerregaard, Peter
Brage, Søren
Pedersen, Oluf
Moltke, Ida
Grarup, Niels
Al-Hasani, Hadi
Albrechtsen, Anders
Jørgensen, Marit E.
Hansen, Torben

Contributor: SpringerLink (Online service)

DOI: 10.1007/s00125-021-05461-z

URN: urn:nbn:de:101:1-2021061419073499045765

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:51 AM CEST

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Associated

Schnurr, Theresia M.
Jørsboe, Emil
Chadt, Alexandra
Dahl-Petersen, Inger K.
Kristensen, Jonas M.
Wojtaszewski, Jørgen F. P.
Springer, Christian
Bjerregaard, Peter
Brage, Søren
Pedersen, Oluf
Moltke, Ida
Grarup, Niels
Al-Hasani, Hadi
Albrechtsen, Anders
Jørgensen, Marit E.
Hansen, Torben
SpringerLink (Online service)

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A proof-of-concept study to evaluate the efficacy and safety of BTI320 on post-prandial hyperglycaemia in Chinese subjects with pre-diabetes

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Physical activity attenuates postprandial hyperglycaemia in homozygous TBC1D4 loss-of-function mutation carriers

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Other Objects (12)

Dual A1 and A2A adenosine receptor antagonists, methoxy substituted 2-benzylidene-1-indanone, suppresses intestinal postprandial glucose and attenuates hyperglycaemia in fructose-streptozotocin diabetic rats

A homozygous loss-of-function mutation leading to CYBC1 deficiency causes chronic granulomatous disease

Homozygous loss-of-function mutations in MNS1 cause laterality defects and likely male infertility

A homozygous loss-of-function variant in BICD2 is associated with lissencephaly and cerebellar hypoplasia

Homozygous loss-of-function variants in FILIP1 cause autosomal recessive arthrogryposis multiplex congenita with microcephaly

SIRT1 activation attenuates α cell hyperplasia, hyperglucagonaemia and hyperglycaemia in STZ-diabetic mice

Hemi- and homozygous loss-of-function mutations in DSG2 (desmoglein-2) cause recessive arrhythmogenic cardiomyopathy with an early onset

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A proof-of-concept study to evaluate the efficacy and safety of BTI320 on post-prandial hyperglycaemia in Chinese subjects with pre-diabetes

Discriminatory ability of anthropometric measurements of central fat distribution for prediction of post-prandial hyperglycaemia in patients with normal fasting glucose: the DICAMANO Study

Dual A1 and A2A adenosine receptor antagonists, methoxy substituted 2-benzylidene-1-indanone, suppresses intestinal postprandial glucose and attenuates hyperglycaemia in fructose-streptozotocin diabetic rats

A homozygous loss-of-function mutation leading to CYBC1 deficiency causes chronic granulomatous disease

Homozygous loss-of-function mutations in MNS1 cause laterality defects and likely male infertility

A homozygous loss-of-function variant in BICD2 is associated with lissencephaly and cerebellar hypoplasia

Homozygous loss-of-function variants in FILIP1 cause autosomal recessive arthrogryposis multiplex congenita with microcephaly

SIRT1 activation attenuates α cell hyperplasia, hyperglucagonaemia and hyperglycaemia in STZ-diabetic mice

Hemi- and homozygous loss-of-function mutations in DSG2 (desmoglein-2) cause recessive arrhythmogenic cardiomyopathy with an early onset

Acute metabolic actions of the major polyphenols in chamomile: an in vitro mechanistic study on their potential to attenuate postprandial hyperglycaemia

Postprandial metabolic changes in healthy males and in subjects homozygous for GWAS-identified variants at lipid metabolism loci

Prevalence of postprandial hyperglycaemia in basal insulin-treated patients with type 2 diabetes mellitus with controlled fasting glycaemia and elevated glycosylated haemoglobin

A proof-of-concept study to evaluate the efficacy and safety of BTI320 on post-prandial hyperglycaemia in Chinese subjects with pre-diabetes

Discriminatory ability of anthropometric measurements of central fat distribution for prediction of post-prandial hyperglycaemia in patients with normal fasting glucose: the DICAMANO Study

Dual A1 and A2A adenosine receptor antagonists, methoxy substituted 2-benzylidene-1-indanone, suppresses intestinal postprandial glucose and attenuates hyperglycaemia in fructose-streptozotocin diabetic rats

A homozygous loss-of-function mutation leading to CYBC1 deficiency causes chronic granulomatous disease

Homozygous loss-of-function mutations in MNS1 cause laterality defects and likely male infertility

A homozygous loss-of-function variant in BICD2 is associated with lissencephaly and cerebellar hypoplasia

Homozygous loss-of-function variants in FILIP1 cause autosomal recessive arthrogryposis multiplex congenita with microcephaly

SIRT1 activation attenuates α cell hyperplasia, hyperglucagonaemia and hyperglycaemia in STZ-diabetic mice

Hemi- and homozygous loss-of-function mutations in DSG2 (desmoglein-2) cause recessive arrhythmogenic cardiomyopathy with an early onset

Acute metabolic actions of the major polyphenols in chamomile: an in vitro mechanistic study on their potential to attenuate postprandial hyperglycaemia

Postprandial metabolic changes in healthy males and in subjects homozygous for GWAS-identified variants at lipid metabolism loci

Prevalence of postprandial hyperglycaemia in basal insulin-treated patients with type 2 diabetes mellitus with controlled fasting glycaemia and elevated glycosylated haemoglobin

A proof-of-concept study to evaluate the efficacy and safety of BTI320 on post-prandial hyperglycaemia in Chinese subjects with pre-diabetes

Discriminatory ability of anthropometric measurements of central fat distribution for prediction of post-prandial hyperglycaemia in patients with normal fasting glucose: the DICAMANO Study

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