- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1750-1326
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Mutations in LRRK2 potentiate age-related impairment of autophagic flux ; volume:10 ; number:1 ; day:11 ; month:7 ; year:2015 ; pages:1-14 ; date:12.2015
Molecular neurodegeneration ; 10, Heft 1 (11.7.2015), 1-14, 12.2015
- Creator
-
Saha, Shamol
Ash, Peter E. A.
Gowda, Vivek
Liu, Liqun
Shirihai, Orian
Wolozin, Benjamin
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1186/s13024-015-0022-y
- URN
-
urn:nbn:de:101:1-2021090122435219321960
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:31 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Saha, Shamol
- Ash, Peter E. A.
- Gowda, Vivek
- Liu, Liqun
- Shirihai, Orian
- Wolozin, Benjamin
- SpringerLink (Online service)
Other Objects (12)
The tricyclic antidepressant clomipramine inhibits neuronal autophagic flux
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Celecoxib alleviates nonalcoholic fatty liver disease by restoring autophagic flux
Metformin ameliorates valve interstitial cell calcification by promoting autophagic flux
Interaction of TBC1D9B with Mammalian ATG8 Homologues Regulates Autophagic Flux
Squaramide-based synthetic chloride transporters activate TFEB but block autophagic flux
Exhaustion of mitochondrial and autophagic reserve may contribute to the development of LRRK2 G2019S -Parkinson’s disease
Ursodeoxycholic acid induces sarcopenia associated with decreased protein synthesis and autophagic flux
Disturbed intracellular folate homeostasis impairs autophagic flux and increases hepatocytic lipid accumulation
Autophagic flux inhibition enhances cytotoxicity of the receptor tyrosine kinase inhibitor ponatinib
Three-dimensional tumor cell growth stimulates autophagic flux and recapitulates chemotherapy resistance
Knockdown ATG4C inhibits gliomas progression and promotes temozolomide chemosensitivity by suppressing autophagic flux
The tricyclic antidepressant clomipramine inhibits neuronal autophagic flux
Rab7b modulates autophagic flux by interacting with Atg4B
Celecoxib alleviates nonalcoholic fatty liver disease by restoring autophagic flux
Metformin ameliorates valve interstitial cell calcification by promoting autophagic flux
Interaction of TBC1D9B with Mammalian ATG8 Homologues Regulates Autophagic Flux
Squaramide-based synthetic chloride transporters activate TFEB but block autophagic flux
Exhaustion of mitochondrial and autophagic reserve may contribute to the development of LRRK2 G2019S -Parkinson’s disease
Ursodeoxycholic acid induces sarcopenia associated with decreased protein synthesis and autophagic flux
Disturbed intracellular folate homeostasis impairs autophagic flux and increases hepatocytic lipid accumulation
Autophagic flux inhibition enhances cytotoxicity of the receptor tyrosine kinase inhibitor ponatinib
Three-dimensional tumor cell growth stimulates autophagic flux and recapitulates chemotherapy resistance
Knockdown ATG4C inhibits gliomas progression and promotes temozolomide chemosensitivity by suppressing autophagic flux
The tricyclic antidepressant clomipramine inhibits neuronal autophagic flux
Rab7b modulates autophagic flux by interacting with Atg4B
Celecoxib alleviates nonalcoholic fatty liver disease by restoring autophagic flux
Metformin ameliorates valve interstitial cell calcification by promoting autophagic flux
Interaction of TBC1D9B with Mammalian ATG8 Homologues Regulates Autophagic Flux
Squaramide-based synthetic chloride transporters activate TFEB but block autophagic flux
Exhaustion of mitochondrial and autophagic reserve may contribute to the development of LRRK2 G2019S -Parkinson’s disease
Ursodeoxycholic acid induces sarcopenia associated with decreased protein synthesis and autophagic flux
Disturbed intracellular folate homeostasis impairs autophagic flux and increases hepatocytic lipid accumulation
Autophagic flux inhibition enhances cytotoxicity of the receptor tyrosine kinase inhibitor ponatinib
Three-dimensional tumor cell growth stimulates autophagic flux and recapitulates chemotherapy resistance