- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1758-9193
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Dystrophic neurites express C9orf72 in Alzheimer's disease brains ; volume:4 ; number:4 ; day:16 ; month:8 ; year:2012 ; pages:1-13 ; date:8.2012
Alzheimer's research & therapy ; 4, Heft 4 (16.8.2012), 1-13, 8.2012
- Creator
-
Satoh, Jun-ichi
- Contributor
-
Tabunoki, Hiroko
Ishida, Tsuyoshi
Saito, Yuko
Arima, Kunimasa
SpringerLink (Online service)
- DOI
-
10.1186/alzrt136
- URN
-
urn:nbn:de:1111-2016073131409
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:54 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Satoh, Jun-ichi
- Tabunoki, Hiroko
- Ishida, Tsuyoshi
- Saito, Yuko
- Arima, Kunimasa
- SpringerLink (Online service)
Other Objects (12)
Sequential formation of different layers of dystrophic neurites in Alzheimer’s brains
Accumulation of saposin in dystrophic neurites is linked to impaired lysosomal functions in Alzheimer’s disease brains
Advanced Immunolabeling Method for Optical Volumetric Imaging Reveals Dystrophic Neurites of Dopaminergic Neurons in Alzheimer’s Disease Mouse Brain
Correction: Advanced Immunolabeling Method for Optical Volumetric Imaging Reveals Dystrophic Neurites of Dopaminergic Neurons in Alzheimer’s Disease Mouse Brain
Pathological assessments for the presence of hexanucleotide repeat expansions in C9ORF72 in Alzheimer’s disease
Abnormal accumulation of extracellular vesicles in hippocampal dystrophic axons and regulation by the primary cilia in Alzheimer’s disease
The Alzheimer’s β-secretase BACE1 localizes to normal presynaptic terminals and to dystrophic presynaptic terminals surrounding amyloid plaques
Expansion of C9ORF72 in amyotrophic lateral sclerosis correlates with brain-computer interface performance
Dystrophic (senescent) rather than activated microglial cells are associated with tau pathology and likely precede neurodegeneration in Alzheimer’s disease
STAT3 targeting in dystrophic epidermolysis bullosa
Ion channel impairments in dystrophic cardiomyocytes
C9ORF72 patient-derived endothelial cells drive blood-brain barrier disruption and contribute to neurotoxicity
Sequential formation of different layers of dystrophic neurites in Alzheimer’s brains
Accumulation of saposin in dystrophic neurites is linked to impaired lysosomal functions in Alzheimer’s disease brains
Advanced Immunolabeling Method for Optical Volumetric Imaging Reveals Dystrophic Neurites of Dopaminergic Neurons in Alzheimer’s Disease Mouse Brain
Correction: Advanced Immunolabeling Method for Optical Volumetric Imaging Reveals Dystrophic Neurites of Dopaminergic Neurons in Alzheimer’s Disease Mouse Brain
Pathological assessments for the presence of hexanucleotide repeat expansions in C9ORF72 in Alzheimer’s disease
Abnormal accumulation of extracellular vesicles in hippocampal dystrophic axons and regulation by the primary cilia in Alzheimer’s disease
The Alzheimer’s β-secretase BACE1 localizes to normal presynaptic terminals and to dystrophic presynaptic terminals surrounding amyloid plaques
Expansion of C9ORF72 in amyotrophic lateral sclerosis correlates with brain-computer interface performance
Dystrophic (senescent) rather than activated microglial cells are associated with tau pathology and likely precede neurodegeneration in Alzheimer’s disease
STAT3 targeting in dystrophic epidermolysis bullosa
Ion channel impairments in dystrophic cardiomyocytes
C9ORF72 patient-derived endothelial cells drive blood-brain barrier disruption and contribute to neurotoxicity
Sequential formation of different layers of dystrophic neurites in Alzheimer’s brains
Accumulation of saposin in dystrophic neurites is linked to impaired lysosomal functions in Alzheimer’s disease brains
Advanced Immunolabeling Method for Optical Volumetric Imaging Reveals Dystrophic Neurites of Dopaminergic Neurons in Alzheimer’s Disease Mouse Brain
Correction: Advanced Immunolabeling Method for Optical Volumetric Imaging Reveals Dystrophic Neurites of Dopaminergic Neurons in Alzheimer’s Disease Mouse Brain
Pathological assessments for the presence of hexanucleotide repeat expansions in C9ORF72 in Alzheimer’s disease
Abnormal accumulation of extracellular vesicles in hippocampal dystrophic axons and regulation by the primary cilia in Alzheimer’s disease
The Alzheimer’s β-secretase BACE1 localizes to normal presynaptic terminals and to dystrophic presynaptic terminals surrounding amyloid plaques
Expansion of C9ORF72 in amyotrophic lateral sclerosis correlates with brain-computer interface performance
Dystrophic (senescent) rather than activated microglial cells are associated with tau pathology and likely precede neurodegeneration in Alzheimer’s disease
STAT3 targeting in dystrophic epidermolysis bullosa
Ion channel impairments in dystrophic cardiomyocytes