Dual therapeutic targeting of MYC and JUNB transcriptional programs for enhanced anti-myeloma activity
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Dual therapeutic targeting of MYC and JUNB transcriptional programs for enhanced anti-myeloma activity ; volume:14 ; number:1 ; day:19 ; month:8 ; year:2024 ; pages:1-12 ; date:12.2024
Blood cancer journal ; 14, Heft 1 (19.8.2024), 1-12, 12.2024
- Creator
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Lind, Judith
Aksoy, Osman
Prchal-Murphy, Michaela
Fan, Fengjuan
Fulciniti, Mariateresa
Stoiber, Dagmar
Bakiri, Latifa
Wagner, Erwin F.
Zwickl-Traxler, Elisabeth
Sattler, Martin
Kollmann, Karoline
Vallet, Sonia
Podar, Klaus
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41408-024-01117-4
- URN
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urn:nbn:de:101:1-2411052116022.479251149434
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:38 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Lind, Judith
- Aksoy, Osman
- Prchal-Murphy, Michaela
- Fan, Fengjuan
- Fulciniti, Mariateresa
- Stoiber, Dagmar
- Bakiri, Latifa
- Wagner, Erwin F.
- Zwickl-Traxler, Elisabeth
- Sattler, Martin
- Kollmann, Karoline
- Vallet, Sonia
- Podar, Klaus
- SpringerLink (Online service)
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Recent advances in searching c-Myc transcriptional cofactors during tumorigenesis
Characterization of MYC Rearrangements in Multiple Myeloma: an Optical Genome Mapping Approach
The OTUD6B‐LIN28B‐MYC axis determines the proliferative state in multiple myeloma
MINCR is a MYC-induced lncRNA able to modulate MYC's transcriptional network in Burkitt lymphoma cells
Novel dual-targeting c-Myc inhibitor D347-2761 represses myeloma growth via blocking c-Myc/Max heterodimerization and disturbing its stability
Targeting MYC in high risk medulloblastoma
Targeting c-MYC with T-Cells
Transcriptional programs: Modelling higher order structure in transcriptional control
Glutaminase inhibition in multiple myeloma induces apoptosis via MYC degradation
Targeting phosphoglycerate dehydrogenase in multiple myeloma
Targeting GPRC5D for multiple myeloma therapy
BRNI: Modular analysis of transcriptional regulatory programs
Recent advances in searching c-Myc transcriptional cofactors during tumorigenesis
Characterization of MYC Rearrangements in Multiple Myeloma: an Optical Genome Mapping Approach
The OTUD6B‐LIN28B‐MYC axis determines the proliferative state in multiple myeloma
MINCR is a MYC-induced lncRNA able to modulate MYC's transcriptional network in Burkitt lymphoma cells
Novel dual-targeting c-Myc inhibitor D347-2761 represses myeloma growth via blocking c-Myc/Max heterodimerization and disturbing its stability
Targeting MYC in high risk medulloblastoma
Targeting c-MYC with T-Cells
Transcriptional programs: Modelling higher order structure in transcriptional control
Glutaminase inhibition in multiple myeloma induces apoptosis via MYC degradation
Targeting phosphoglycerate dehydrogenase in multiple myeloma
Targeting GPRC5D for multiple myeloma therapy
BRNI: Modular analysis of transcriptional regulatory programs
Recent advances in searching c-Myc transcriptional cofactors during tumorigenesis
Characterization of MYC Rearrangements in Multiple Myeloma: an Optical Genome Mapping Approach
The OTUD6B‐LIN28B‐MYC axis determines the proliferative state in multiple myeloma