Dipeptide repeat proteins are present in the p62 positive inclusions in patients with frontotemporal lobar degeneration and motor neurone disease associated with expansions in C9ORF72
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2051-5960
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Dipeptide repeat proteins are present in the p62 positive inclusions in patients with frontotemporal lobar degeneration and motor neurone disease associated with expansions in C9ORF72 ; volume:1 ; number:1 ; day:14 ; month:10 ; year:2013 ; pages:1-13 ; date:12.2013
Acta Neuropathologica Communications ; 1, Heft 1 (14.10.2013), 1-13, 12.2013
- Creator
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Mann, David MA
- Contributor
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Rollinson, Sara
Robinson, Andrew
Bennion Callister, Janis
Thompson, Jennifer C.
Snowden, Julie S.
Gendron, Tania
Petrucelli, Leonard
Masuda-Suzukake, Masami
Hasegawa, Masato
Davidson, Yvonne
Pickering-Brown, Stuart
SpringerLink (Online service)
- DOI
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10.1186/2051-5960-1-68
- URN
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urn:nbn:de:1111-2016052412291
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:44 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Mann, David MA
- Rollinson, Sara
- Robinson, Andrew
- Bennion Callister, Janis
- Thompson, Jennifer C.
- Snowden, Julie S.
- Gendron, Tania
- Petrucelli, Leonard
- Masuda-Suzukake, Masami
- Hasegawa, Masato
- Davidson, Yvonne
- Pickering-Brown, Stuart
- SpringerLink (Online service)
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Peripheral inflammatory markers and clinical correlations in patients with frontotemporal lobar degeneration with and without the C9orf72 repeat expansion
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Bidirectional nucleolar dysfunction in C9orf72 frontotemporal lobar degeneration
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Characterization of the C9orf72 dipeptide repeat proteins in frontotemporal dementia and amyotrophic lateral sclerosis
Delusions in frontotemporal lobar degeneration
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Apraxia phenotypes and frontotemporal lobar degeneration
FDG-PET underscores the key role of the thalamus in frontotemporal lobar degeneration caused by C9ORF72 mutations
Heterogeneous ribonuclear protein A3 (hnRNP A3) is present in dipeptide repeat protein containing inclusions in Frontotemporal Lobar Degeneration and Motor Neurone disease associated with expansions in C9orf72 gene
TAF15 amyloid filaments in frontotemporal lobar degeneration
Progranulin in frontotemporal lobar degeneration and neuroinflammation
Peripheral inflammatory markers and clinical correlations in patients with frontotemporal lobar degeneration with and without the C9orf72 repeat expansion
Metabolomic changes associated with frontotemporal lobar degeneration syndromes
Bidirectional nucleolar dysfunction in C9orf72 frontotemporal lobar degeneration
Brain distribution of dipeptide repeat proteins in frontotemporal lobar degeneration and motor neurone disease associated with expansions in C9ORF72
Characterization of the C9orf72 dipeptide repeat proteins in frontotemporal dementia and amyotrophic lateral sclerosis
Delusions in frontotemporal lobar degeneration
Frontotemporal Lobar Degeneration: A Clinical Approach
Apraxia phenotypes and frontotemporal lobar degeneration
FDG-PET underscores the key role of the thalamus in frontotemporal lobar degeneration caused by C9ORF72 mutations
Heterogeneous ribonuclear protein A3 (hnRNP A3) is present in dipeptide repeat protein containing inclusions in Frontotemporal Lobar Degeneration and Motor Neurone disease associated with expansions in C9orf72 gene
TAF15 amyloid filaments in frontotemporal lobar degeneration
Progranulin in frontotemporal lobar degeneration and neuroinflammation
Peripheral inflammatory markers and clinical correlations in patients with frontotemporal lobar degeneration with and without the C9orf72 repeat expansion