- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1465-542X
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Akt is required for Stat5 activation and mammary differentiation ; volume:12 ; number:5 ; day:17 ; month:9 ; year:2010 ; pages:1-14 ; date:10.2010
Breast cancer research ; 12, Heft 5 (17.9.2010), 1-14, 10.2010
- Creator
-
Chen, Chien-Chung
- Contributor
-
Boxer, Robert B.
Stairs, Douglas B.
Portocarrero, Carla P.
Horton, Rachel H.
Alvarez, James V.
Birnbaum, Morris J.
Chodosh, Lewis A.
SpringerLink (Online service)
- DOI
-
10.1186/bcr2640
- URN
-
urn:nbn:de:1111-201603248561
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:54 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Chen, Chien-Chung
- Boxer, Robert B.
- Stairs, Douglas B.
- Portocarrero, Carla P.
- Horton, Rachel H.
- Alvarez, James V.
- Birnbaum, Morris J.
- Chodosh, Lewis A.
- SpringerLink (Online service)
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Miz1 Deficiency in the Mammary Gland Causes a Lactation Defect by Attenuated Stat5 Expression and Phosphorylation
A co-culture genome-wide RNAi screen with mammary epithelial cells reveals transmembrane signals required for growth and differentiation
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The glucose transporter GLUT1 is required for ErbB2-induced mammary tumorigenesis
O-GlcNAcylation of STAT5 controls tyrosine phosphorylation and oncogenic transcription in STAT5-dependent malignancies
Bortezomib inhibits STAT5-dependent degradation of LEF-1, inducing granulocytic differentiation in congenital neutropenia CD34+ cells
PTHrP induces STAT5 activation, secretory differentiation and accelerates mammary tumor development
PI3Kγ kinase activity is required for optimal T‐cell activation and differentiation
Emancipation from transcriptional latency: unphosphorylated STAT5 as guardian of hematopoietic differentiation
TNFAIP3 is required for FGFR1 activation-promoted proliferation and tumorigenesis of premalignant DCIS.COM human mammary epithelial cells
Activation function 1 of progesterone receptor is required for mammary development and regulation of RANKL during pregnancy
Miz1 Deficiency in the Mammary Gland Causes a Lactation Defect by Attenuated Stat5 Expression and Phosphorylation
A co-culture genome-wide RNAi screen with mammary epithelial cells reveals transmembrane signals required for growth and differentiation
Fibulin-2 is required for basement membrane integrity of mammary epithelium
ErbB3 is required for ductal morphogenesis in the mouse mammary gland
The glucose transporter GLUT1 is required for ErbB2-induced mammary tumorigenesis
O-GlcNAcylation of STAT5 controls tyrosine phosphorylation and oncogenic transcription in STAT5-dependent malignancies
Bortezomib inhibits STAT5-dependent degradation of LEF-1, inducing granulocytic differentiation in congenital neutropenia CD34+ cells
PTHrP induces STAT5 activation, secretory differentiation and accelerates mammary tumor development
PI3Kγ kinase activity is required for optimal T‐cell activation and differentiation
Emancipation from transcriptional latency: unphosphorylated STAT5 as guardian of hematopoietic differentiation
TNFAIP3 is required for FGFR1 activation-promoted proliferation and tumorigenesis of premalignant DCIS.COM human mammary epithelial cells
Activation function 1 of progesterone receptor is required for mammary development and regulation of RANKL during pregnancy
Miz1 Deficiency in the Mammary Gland Causes a Lactation Defect by Attenuated Stat5 Expression and Phosphorylation
A co-culture genome-wide RNAi screen with mammary epithelial cells reveals transmembrane signals required for growth and differentiation
Fibulin-2 is required for basement membrane integrity of mammary epithelium
ErbB3 is required for ductal morphogenesis in the mouse mammary gland
The glucose transporter GLUT1 is required for ErbB2-induced mammary tumorigenesis
O-GlcNAcylation of STAT5 controls tyrosine phosphorylation and oncogenic transcription in STAT5-dependent malignancies