Amyloid and tau PET-positive cognitively unimpaired individuals are at high risk for future cognitive decline
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1546-170X
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Amyloid and tau PET-positive cognitively unimpaired individuals are at high risk for future cognitive decline ; volume:28 ; number:11 ; day:10 ; month:11 ; year:2022 ; pages:2381-2387 ; date:11.2022
Nature medicine ; 28, Heft 11 (10.11.2022), 2381-2387, 11.2022
- Creator
-
Ossenkoppele, Rik
Pichet Binette, Alexa
Groot, Colin
Smith, Ruben
Strandberg, Olof
Palmqvist, Sebastian
Stomrud, Erik
Tideman, Pontus
Ohlsson, Tomas
Jögi, Jonas
Johnson, Keith
Sperling, Reisa
Dore, Vincent
Masters, Colin L.
Rowe, Christopher
Visser, Denise
Berckel, Bart N. M. van
Flier, Wiesje M. van der
Baker, Suzanne
Jagust, William J.
Wiste, Heather J.
Petersen, Ronald C.
Jack, Clifford R.
Hansson, Oskar
- Contributor
-
SpringerLink (Online service)
- DOI
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10.1038/s41591-022-02049-x
- URN
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urn:nbn:de:101:1-2023013021121993662770
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
07.08.2024, 1:26 AM CEST
Data provider
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Associated
- Ossenkoppele, Rik
- Pichet Binette, Alexa
- Groot, Colin
- Smith, Ruben
- Strandberg, Olof
- Palmqvist, Sebastian
- Stomrud, Erik
- Tideman, Pontus
- Ohlsson, Tomas
- Jögi, Jonas
- Johnson, Keith
- Sperling, Reisa
- Dore, Vincent
- Masters, Colin L.
- Rowe, Christopher
- Visser, Denise
- Berckel, Bart N. M. van
- Flier, Wiesje M. van der
- Baker, Suzanne
- Jagust, William J.
- Wiste, Heather J.
- Petersen, Ronald C.
- Jack, Clifford R.
- Hansson, Oskar
- SpringerLink (Online service)
Other Objects (12)
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Cognitively normal women with Alzheimer’s disease proteinopathy show relative preservation of memory but not of hippocampal volume
Amyloid-associated increases in soluble tau relate to tau aggregation rates and cognitive decline in early Alzheimer’s disease
Cerebrospinal fluid N-224 tau helps discriminate Alzheimer’s disease from subjective cognitive decline and other dementias
The age-related effect on cognitive performance in cognitively healthy elderly is mainly caused by underlying AD pathology or cerebrovascular lesions: implications for cutoffs regarding cognitive impairment
Effects of certain pre-analytical factors on the performance of plasma phospho-tau217
Antibodies against phosphorylcholine are not altered in plasma of patients with Alzheimer’s disease
Characterization of degradation and heterozygote balance by simulation of the forensic DNA analysis process
Specific associations between plasma biomarkers and postmortem amyloid plaque and tau tangle loads
Axonal degeneration and amyloid pathology predict cognitive decline beyond cortical atrophy
Cerebro-spinal fluid biomarker levels: phosphorylated tau (T) and total tau (N) as markers for rate of progression in Alzheimer’s disease
Soluble P‐tau217 reflects amyloid and tau pathology and mediates the association of amyloid with tau
Towards a unified protocol for handling of CSF before β-amyloid measurements
Cognitively normal women with Alzheimer’s disease proteinopathy show relative preservation of memory but not of hippocampal volume
Amyloid-associated increases in soluble tau relate to tau aggregation rates and cognitive decline in early Alzheimer’s disease
Cerebrospinal fluid N-224 tau helps discriminate Alzheimer’s disease from subjective cognitive decline and other dementias
The age-related effect on cognitive performance in cognitively healthy elderly is mainly caused by underlying AD pathology or cerebrovascular lesions: implications for cutoffs regarding cognitive impairment
Effects of certain pre-analytical factors on the performance of plasma phospho-tau217
Antibodies against phosphorylcholine are not altered in plasma of patients with Alzheimer’s disease
Characterization of degradation and heterozygote balance by simulation of the forensic DNA analysis process
Specific associations between plasma biomarkers and postmortem amyloid plaque and tau tangle loads
Axonal degeneration and amyloid pathology predict cognitive decline beyond cortical atrophy
Cerebro-spinal fluid biomarker levels: phosphorylated tau (T) and total tau (N) as markers for rate of progression in Alzheimer’s disease
Soluble P‐tau217 reflects amyloid and tau pathology and mediates the association of amyloid with tau
Towards a unified protocol for handling of CSF before β-amyloid measurements
Cognitively normal women with Alzheimer’s disease proteinopathy show relative preservation of memory but not of hippocampal volume
Amyloid-associated increases in soluble tau relate to tau aggregation rates and cognitive decline in early Alzheimer’s disease
Cerebrospinal fluid N-224 tau helps discriminate Alzheimer’s disease from subjective cognitive decline and other dementias
The age-related effect on cognitive performance in cognitively healthy elderly is mainly caused by underlying AD pathology or cerebrovascular lesions: implications for cutoffs regarding cognitive impairment
Effects of certain pre-analytical factors on the performance of plasma phospho-tau217
Antibodies against phosphorylcholine are not altered in plasma of patients with Alzheimer’s disease
Characterization of degradation and heterozygote balance by simulation of the forensic DNA analysis process