Role of oxidative stress and inflammation-related signaling pathways in doxorubicin-induced cardiomyopathy
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Role of oxidative stress and inflammation-related signaling pathways in doxorubicin-induced cardiomyopathy ; volume:21 ; number:1 ; day:14 ; month:3 ; year:2023 ; pages:1-20 ; date:12.2023
Cell communication and signaling ; 21, Heft 1 (14.3.2023), 1-20, 12.2023
- Creator
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Shi, Saixian
Chen, Ye
Luo, Zhijian
Nie, Guojun
Dai, Yan
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s12964-023-01077-5
- URN
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urn:nbn:de:101:1-2023080421200208518651
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 11:01 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Shi, Saixian
- Chen, Ye
- Luo, Zhijian
- Nie, Guojun
- Dai, Yan
- SpringerLink (Online service)
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Calsyntenin-1 Promotes Doxorubicin-induced Dilated Cardiomyopathy in Rats
Recent Progress in Microfiber-Optic Sensors
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Embryonic stem cell-derived cardiomyocytes for the treatment of doxorubicin-induced cardiomyopathy
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Major vault protein attenuates cardiomyocyte injury in doxorubicin-induced cardiomyopathy through activating AKT
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Fabrication of apigenin and adenosine-loaded nanoparticles against doxorubicin-induced myocardial infarction by reducing inflammation and oxidative stress
The ill-posedness of the (non-) periodic traveling wave solution for the deformed continuous Heisenberg spin equation
Co-crosslinking strategy for dual functionalization of small magnetic nanoparticles with redox probes and biological probes
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MST1 mediates doxorubicin-induced cardiomyopathy by SIRT3 downregulation
Calsyntenin-1 Promotes Doxorubicin-induced Dilated Cardiomyopathy in Rats
Recent Progress in Microfiber-Optic Sensors
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Embryonic stem cell-derived cardiomyocytes for the treatment of doxorubicin-induced cardiomyopathy
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Major vault protein attenuates cardiomyocyte injury in doxorubicin-induced cardiomyopathy through activating AKT