Hochschulschrift
TLR/MyD88 signaling in B cells suppresses T cell-mediated CNS autoimmunity
- Alternative title
-
Der TLR/MyD88 Signalweg in B Zellen supprimiert T-Zell -vermittelte Autoimmunität des ZNS
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
Berlin, Technische Universtität Berlin, Diss., 2011
- Classification
-
Medizin, Gesundheit
- Event
-
Veröffentlichung
- (where)
-
Berlin
- (who)
-
Universitätsbibliothek der Technischen Universität Berlin
- (when)
-
2012
- Creator
-
Lampropoulou, Vasiliki
- Contributor
-
Lauster, Roland
- URN
-
urn:nbn:de:kobv:83-opus-35089
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:20 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Object type
- Hochschulschrift
Associated
- Lampropoulou, Vasiliki
- Lauster, Roland
- Universitätsbibliothek der Technischen Universität Berlin
Time of origin
- 2012
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Stability and plasticity of IL-17 expression in TH17 cells
Improvement of adoptive T cell therapy by T cell specific targeting of T cell inhibitory receptors instead of systemic antibody treatment
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Massenspektrometrische Identifizierung posttranslationaler Modifizierungen mit monomerem N-Acetylglukosamin an Proteasomen
CD40 stimulation activates ‘post-activated’ B Cells which are hyporesponsive to B Cell receptor and toll-like receptor 9 stimulation in autoimmunity
Expansion of embryonic stem cells in 3D-bioreactors
GDF3: a new player in cancer stem cell biology
Stability and plasticity of IL-17 expression in TH17 cells
Improvement of adoptive T cell therapy by T cell specific targeting of T cell inhibitory receptors instead of systemic antibody treatment
In vitro nephrogenesis from human pluripotent stem cells
Characterization of plasma cell subsets in the human bone marrow
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Analyse zum Einfluss der Hypoxie induzierbaren Faktoren HIF-1 und HIF-2 auf die Angiogenese von Endothelzellen
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