Loss of the proteasomal deubiquitinase USP14 induces growth defects and a senescence phenotype in colorectal cancer cells
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
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1 Online-Ressource.
- Sprache
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Englisch
- Erschienen in
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Loss of the proteasomal deubiquitinase USP14 induces growth defects and a senescence phenotype in colorectal cancer cells ; volume:14 ; number:1 ; day:6 ; month:6 ; year:2024 ; pages:1-14 ; date:12.2024
Scientific reports ; 14, Heft 1 (6.6.2024), 1-14, 12.2024
- Urheber
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Gubat, Johannes
Sjöstrand, Linda
Selvaraju, Karthik
Telli, Kübra
D’Arcy, Pádraig
- Beteiligte Personen und Organisationen
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SpringerLink (Online service)
- DOI
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10.1038/s41598-024-63791-5
- URN
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urn:nbn:de:101:1-2408132121131.720952325919
- Rechteinformation
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
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14.08.2025, 10:47 MESZ
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Beteiligte
- Gubat, Johannes
- Sjöstrand, Linda
- Selvaraju, Karthik
- Telli, Kübra
- D’Arcy, Pádraig
- SpringerLink (Online service)
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Cilia defects upon loss of WDR4 are linked to proteasomal hyperactivity and ubiquitin shortage
Urokinase receptor in regulation of cellular senescence and DNA damage response : role for the ubiquitin-proteasomal system
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Metal-based proteasomal deubiquitinase inhibitors as potential anticancer agents
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Urokinase receptor in regulation of cellular senescence and DNA damage response : role for the ubiquitin-proteasomal system
Small-molecule MDM2 antagonists attenuate the senescence-associated secretory phenotype
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