XPO1 Gene Therapy Attenuates Cardiac Dysfunction in Rats with Chronic Induced Myocardial Infarction

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1937-5395

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: XPO1 Gene Therapy Attenuates Cardiac Dysfunction in Rats with Chronic Induced Myocardial Infarction ; day:25 ; month:11 ; year:2019 ; pages:1-8
Journal of cardiovascular translational research ; (25.11.2019), 1-8

Creator: García-Manzanares, María
Tarazón, Estefanía
Ortega, Ana
Gil-Cayuela, Carolina
Martínez-Dolz, Luis
González-Juanatey, José Ramón
Lago, Francisca
Portolés, Manuel
Roselló-Lletí, Esther
Rivera, Miguel

Contributor: SpringerLink (Online service)

DOI: 10.1007/s12265-019-09932-y

URN: urn:nbn:de:101:1-2020041023232909514239

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:46 AM CEST

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Associated

García-Manzanares, María
Tarazón, Estefanía
Ortega, Ana
Gil-Cayuela, Carolina
Martínez-Dolz, Luis
González-Juanatey, José Ramón
Lago, Francisca
Portolés, Manuel
Roselló-Lletí, Esther
Rivera, Miguel
SpringerLink (Online service)

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XPO1 Gene Therapy Attenuates Cardiac Dysfunction in Rats with Chronic Induced Myocardial Infarction

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Other Objects (12)

Zinc Supplementation Attenuates Cardiac Remodeling After Experimental Myocardial Infarction

Roselle attenuates cardiac hypertrophy after myocardial infarction in vivo and in vitro

The role of COX2 deficiency attenuates cardiac damage in acute myocardial infarction

Cardiac-specific overexpression of Ndufs1 ameliorates cardiac dysfunction after myocardial infarction by alleviating mitochondrial dysfunction and apoptosis

Cardiac Shock Wave Therapy Attenuates Cardiomyocyte Apoptosis after Acute Myocardial Infarction in Rats

Cholinergic Stimulation by Pyridostigmine Bromide Before Myocardial Infarction Prevent Cardiac and Autonomic Dysfunction

Dusp6 deficiency attenuates neutrophil-mediated cardiac damage in the acute inflammatory phase of myocardial infarction

Eosinophils improve cardiac function after myocardial infarction

Erythropoietin attenuates cardiac dysfunction by increasing myocardial angiogenesis and inhibiting interstitial fibrosis in diabetic rats

HDAC5 inhibition attenuates ventricular remodeling and cardiac dysfunction

MicroRNA-21 prevents excessive inflammation and cardiac dysfunction after myocardial infarction through targeting KBTBD7

Sfrp1 attenuates TAC-induced cardiac dysfunction by inhibiting Wnt signaling pathway- mediated myocardial apoptosis in mice

Zinc Supplementation Attenuates Cardiac Remodeling After Experimental Myocardial Infarction

Roselle attenuates cardiac hypertrophy after myocardial infarction in vivo and in vitro

The role of COX2 deficiency attenuates cardiac damage in acute myocardial infarction

Cardiac-specific overexpression of Ndufs1 ameliorates cardiac dysfunction after myocardial infarction by alleviating mitochondrial dysfunction and apoptosis

Cardiac Shock Wave Therapy Attenuates Cardiomyocyte Apoptosis after Acute Myocardial Infarction in Rats

Cholinergic Stimulation by Pyridostigmine Bromide Before Myocardial Infarction Prevent Cardiac and Autonomic Dysfunction

Dusp6 deficiency attenuates neutrophil-mediated cardiac damage in the acute inflammatory phase of myocardial infarction

Eosinophils improve cardiac function after myocardial infarction

Erythropoietin attenuates cardiac dysfunction by increasing myocardial angiogenesis and inhibiting interstitial fibrosis in diabetic rats

HDAC5 inhibition attenuates ventricular remodeling and cardiac dysfunction

MicroRNA-21 prevents excessive inflammation and cardiac dysfunction after myocardial infarction through targeting KBTBD7

Sfrp1 attenuates TAC-induced cardiac dysfunction by inhibiting Wnt signaling pathway- mediated myocardial apoptosis in mice

Zinc Supplementation Attenuates Cardiac Remodeling After Experimental Myocardial Infarction

Roselle attenuates cardiac hypertrophy after myocardial infarction in vivo and in vitro

The role of COX2 deficiency attenuates cardiac damage in acute myocardial infarction

Cardiac-specific overexpression of Ndufs1 ameliorates cardiac dysfunction after myocardial infarction by alleviating mitochondrial dysfunction and apoptosis

Cardiac Shock Wave Therapy Attenuates Cardiomyocyte Apoptosis after Acute Myocardial Infarction in Rats

Cholinergic Stimulation by Pyridostigmine Bromide Before Myocardial Infarction Prevent Cardiac and Autonomic Dysfunction

Dusp6 deficiency attenuates neutrophil-mediated cardiac damage in the acute inflammatory phase of myocardial infarction

Eosinophils improve cardiac function after myocardial infarction

Erythropoietin attenuates cardiac dysfunction by increasing myocardial angiogenesis and inhibiting interstitial fibrosis in diabetic rats

HDAC5 inhibition attenuates ventricular remodeling and cardiac dysfunction

MicroRNA-21 prevents excessive inflammation and cardiac dysfunction after myocardial infarction through targeting KBTBD7

Sfrp1 attenuates TAC-induced cardiac dysfunction by inhibiting Wnt signaling pathway- mediated myocardial apoptosis in mice

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