Hochschulschrift
Characterisation of axonal degeneration during autoimune optic neuritis
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
Heidelberg, Universität Heidelberg, Dissertation, 2016
- Schlagwort
-
Optic neuritis
Degeneration
Optic nerve
Pathology
Nerves
- Ereignis
-
Veröffentlichung
- (wo)
-
Heidelberg
- (wer)
-
Universitätsbibliothek Heidelberg
- (wann)
-
2016
- Urheber
- Beteiligte Personen und Organisationen
-
Régnier-Vigouroux, Anne
- DOI
-
10.11588/heidok.00020426
- URN
-
urn:nbn:de:bsz:16-heidok-204262
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
25.03.2025, 13:51 MEZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Objekttyp
- Hochschulschrift
Beteiligte
- Stojic, Aleksandar
- Régnier-Vigouroux, Anne
- Universitätsbibliothek Heidelberg
Entstanden
- 2016
Ähnliche Objekte (12)
Characterisation of axonal degeneration during autoimmune optic neuritis
Progression of RGC degeneration during murine autoimmune optic neuritis
Molecular mechanisms of acute axonal degeneration in the rat optic nerve
Calcium-mediated mechanisms of retinal ganglion cell degeneration during autoimmune optic neuritis
Calcium-mediated mechanisms of retinal ganglion cell degeneration during autoimmune optic neuritis
Axonal degeneration in Parkinson's disease : = Axonale Degeneration beim Parkinson Syndrom
Involvement of N-type voltage dependent calcium channels in axon degeneration during experimental autoimmune optic neuritis
Disease course of clinically isolated optic neuritis
Molecular mechanisms of the axonal degeneration in hereditary neuropathies
In vivo imaging of axonal degeneration and axonal transport in mouse models of multiple sclerosis
Dimethyl fumarate ameliorates Lewis rat experimental autoimmune neuritis and mediates axonal protection
Oligoclonal bands predict multiple sclerosis in children with optic neuritis
Characterisation of axonal degeneration during autoimmune optic neuritis
Progression of RGC degeneration during murine autoimmune optic neuritis
Molecular mechanisms of acute axonal degeneration in the rat optic nerve
Calcium-mediated mechanisms of retinal ganglion cell degeneration during autoimmune optic neuritis
Calcium-mediated mechanisms of retinal ganglion cell degeneration during autoimmune optic neuritis
Axonal degeneration in Parkinson's disease : = Axonale Degeneration beim Parkinson Syndrom
Involvement of N-type voltage dependent calcium channels in axon degeneration during experimental autoimmune optic neuritis
Disease course of clinically isolated optic neuritis
Molecular mechanisms of the axonal degeneration in hereditary neuropathies
In vivo imaging of axonal degeneration and axonal transport in mouse models of multiple sclerosis
Dimethyl fumarate ameliorates Lewis rat experimental autoimmune neuritis and mediates axonal protection
Oligoclonal bands predict multiple sclerosis in children with optic neuritis
Characterisation of axonal degeneration during autoimmune optic neuritis
Progression of RGC degeneration during murine autoimmune optic neuritis
Molecular mechanisms of acute axonal degeneration in the rat optic nerve
Calcium-mediated mechanisms of retinal ganglion cell degeneration during autoimmune optic neuritis
Calcium-mediated mechanisms of retinal ganglion cell degeneration during autoimmune optic neuritis
Axonal degeneration in Parkinson's disease : = Axonale Degeneration beim Parkinson Syndrom
Involvement of N-type voltage dependent calcium channels in axon degeneration during experimental autoimmune optic neuritis
Disease course of clinically isolated optic neuritis
Molecular mechanisms of the axonal degeneration in hereditary neuropathies
In vivo imaging of axonal degeneration and axonal transport in mouse models of multiple sclerosis
Dimethyl fumarate ameliorates Lewis rat experimental autoimmune neuritis and mediates axonal protection