Obesity aggravates acute kidney injury resulting from ischemia and reperfusion in mice
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Obesity aggravates acute kidney injury resulting from ischemia and reperfusion in mice ; volume:14 ; number:1 ; day:29 ; month:4 ; year:2024 ; pages:1-11 ; date:12.2024
Scientific reports ; 14, Heft 1 (29.4.2024), 1-11, 12.2024
- Creator
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Silva, Igor Oliveira da
Menezes, Nicole K. de
Jacobina, Heloisa D.
Parra, Antonio Carlos
Souza, Felipe Lima
Castro, Leticia Cardoso
Roelofs, Joris J. T. H.
Tammaro, Alessandra
Gomes, Samirah Abreu
Sanches, Talita Rojas
Andrade, Lucia
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41598-024-60365-3
- URN
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urn:nbn:de:101:1-2407242106423.425389645992
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:44 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Silva, Igor Oliveira da
- Menezes, Nicole K. de
- Jacobina, Heloisa D.
- Parra, Antonio Carlos
- Souza, Felipe Lima
- Castro, Leticia Cardoso
- Roelofs, Joris J. T. H.
- Tammaro, Alessandra
- Gomes, Samirah Abreu
- Sanches, Talita Rojas
- Andrade, Lucia
- SpringerLink (Online service)
Other Objects (12)
Author Correction: Obesity aggravates acute kidney injury resulting from ischemia and reperfusion in mice
Labile heme aggravates renal inflammation and complement activation after ischemia reperfusion injury
IKK1 aggravates ischemia–reperfusion kidney injury by promoting the differentiation of effector T cells
miR-10a overexpression aggravates renal ischemia–reperfusion injury associated with decreased PIK3CA expression
Mitochondria-derived methylmalonic acid aggravates ischemia–reperfusion injury by activating reactive oxygen species-dependent ferroptosis
Cold Preflush of Porcine Kidney Grafts Prior to Normothermic Machine Perfusion Aggravates Ischemia Reperfusion Injury
Rictor Deficiency Aggravates Hepatic Ischemia/Reperfusion Injury in Mice by Suppressing Autophagy and Regulating MAPK Signaling
Ischemia-induced cleavage of OPA1 at S1 site aggravates mitochondrial fragmentation and reperfusion injury in neurons
Cpg-ODN, a TLR9 Agonist, Aggravates Myocardial Ischemia/Reperfusion Injury by Activation of TLR9-P38 MAPK Signaling
Inhibition of PTEN Activity Aggravates Post Renal Fibrosis in Mice with Ischemia Reperfusion-Induced Acute Kidney Injury
Homozygous Smpd1 deficiency aggravates brain ischemia/ reperfusion injury by mechanisms involving polymorphonuclear neutrophils, whereas heterozygous Smpd1 deficiency protects against mild focal cerebral ischemia
Author Correction: Ischemia-induced cleavage of OPA1 at S1 site aggravates mitochondrial fragmentation and reperfusion injury in neurons
Author Correction: Obesity aggravates acute kidney injury resulting from ischemia and reperfusion in mice
Labile heme aggravates renal inflammation and complement activation after ischemia reperfusion injury
IKK1 aggravates ischemia–reperfusion kidney injury by promoting the differentiation of effector T cells
miR-10a overexpression aggravates renal ischemia–reperfusion injury associated with decreased PIK3CA expression
Mitochondria-derived methylmalonic acid aggravates ischemia–reperfusion injury by activating reactive oxygen species-dependent ferroptosis
Cold Preflush of Porcine Kidney Grafts Prior to Normothermic Machine Perfusion Aggravates Ischemia Reperfusion Injury
Rictor Deficiency Aggravates Hepatic Ischemia/Reperfusion Injury in Mice by Suppressing Autophagy and Regulating MAPK Signaling
Ischemia-induced cleavage of OPA1 at S1 site aggravates mitochondrial fragmentation and reperfusion injury in neurons
Cpg-ODN, a TLR9 Agonist, Aggravates Myocardial Ischemia/Reperfusion Injury by Activation of TLR9-P38 MAPK Signaling
Inhibition of PTEN Activity Aggravates Post Renal Fibrosis in Mice with Ischemia Reperfusion-Induced Acute Kidney Injury
Homozygous Smpd1 deficiency aggravates brain ischemia/ reperfusion injury by mechanisms involving polymorphonuclear neutrophils, whereas heterozygous Smpd1 deficiency protects against mild focal cerebral ischemia
Author Correction: Ischemia-induced cleavage of OPA1 at S1 site aggravates mitochondrial fragmentation and reperfusion injury in neurons
Author Correction: Obesity aggravates acute kidney injury resulting from ischemia and reperfusion in mice
Labile heme aggravates renal inflammation and complement activation after ischemia reperfusion injury
IKK1 aggravates ischemia–reperfusion kidney injury by promoting the differentiation of effector T cells
miR-10a overexpression aggravates renal ischemia–reperfusion injury associated with decreased PIK3CA expression
Mitochondria-derived methylmalonic acid aggravates ischemia–reperfusion injury by activating reactive oxygen species-dependent ferroptosis
Cold Preflush of Porcine Kidney Grafts Prior to Normothermic Machine Perfusion Aggravates Ischemia Reperfusion Injury
Rictor Deficiency Aggravates Hepatic Ischemia/Reperfusion Injury in Mice by Suppressing Autophagy and Regulating MAPK Signaling
Ischemia-induced cleavage of OPA1 at S1 site aggravates mitochondrial fragmentation and reperfusion injury in neurons
Cpg-ODN, a TLR9 Agonist, Aggravates Myocardial Ischemia/Reperfusion Injury by Activation of TLR9-P38 MAPK Signaling
Inhibition of PTEN Activity Aggravates Post Renal Fibrosis in Mice with Ischemia Reperfusion-Induced Acute Kidney Injury
Homozygous Smpd1 deficiency aggravates brain ischemia/ reperfusion injury by mechanisms involving polymorphonuclear neutrophils, whereas heterozygous Smpd1 deficiency protects against mild focal cerebral ischemia