- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1432-0878
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Primary cilia disappear in rat podocytes during glomerular development ; volume:341 ; number:1 ; day:22 ; month:5 ; year:2010 ; pages:197-209 ; date:7.2010
Cell & tissue research ; 341, Heft 1 (22.5.2010), 197-209, 7.2010
- Creator
-
Ichimura, Koichiro
Kurihara, Hidetake
Sakai, Tatsuo
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1007/s00441-010-0983-7
- URN
-
urn:nbn:de:101:1-2021081423115170207310
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:52 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Ichimura, Koichiro
- Kurihara, Hidetake
- Sakai, Tatsuo
- SpringerLink (Online service)
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Regulation of cofilin phosphorylation in glomerular podocytes by testis specific kinase 1 (TESK1)
Primary cilia suppress Ripk3-mediated necroptosis
Possible role of complement factor H in podocytes in clearing glomerular subendothelial immune complex deposits
Evolutionary morphology of podocytes and primary urine-producing apparatus
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Actin dynamics in glomerular podocytes
Primary cultures of glomerular parietal epithelial cells or podocytes with proven origin
Osteocytes and Primary Cilia
Rac1 in podocytes promotes glomerular repair and limits the formation of sclerosis
Primary cilia utilize glycoprotein-dependent adhesion mechanisms to stabilize long-lasting cilia-cilia contacts
Mechanical behavior of primary cilia
FRET-based visualization of cGMP dynamics in glomerular endothelial cells and podocytes
Regulation of cofilin phosphorylation in glomerular podocytes by testis specific kinase 1 (TESK1)
Primary cilia suppress Ripk3-mediated necroptosis
Possible role of complement factor H in podocytes in clearing glomerular subendothelial immune complex deposits
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