Suppression of Homologous Recombination by insulin-like growth factor-1 inhibition sensitizes cancer cells to PARP inhibitors

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1471-2407

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Suppression of Homologous Recombination by insulin-like growth factor-1 inhibition sensitizes cancer cells to PARP inhibitors ; volume:15 ; number:1 ; day:29 ; month:10 ; year:2015 ; pages:1-14 ; date:12.2015
BMC cancer ; 15, Heft 1 (29.10.2015), 1-14, 12.2015

Creator: Amin, Oreekha

Contributor: Beauchamp, Marie-Claude
Nader, Paul Abou
Laskov, Ido
Iqbal, Sanaa
Philip, Charles-André
Yasmeen, Amber
Gotlieb, Walter H.
SpringerLink (Online service)

DOI: 10.1186/s12885-015-1803-y

URN: urn:nbn:de:1111-2016110621621

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:47 AM CEST

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Associated

Amin, Oreekha
Beauchamp, Marie-Claude
Nader, Paul Abou
Laskov, Ido
Iqbal, Sanaa
Philip, Charles-André
Yasmeen, Amber
Gotlieb, Walter H.
SpringerLink (Online service)

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CBL0137 impairs homologous recombination repair and sensitizes high-grade serous ovarian carcinoma to PARP inhibitors

Loss of NFBD1/MDC1 disrupts homologous recombination repair and sensitizes nasopharyngeal carcinoma cells to PARP inhibitors

PLK1 overexpression suppresses homologous recombination and confers cellular sensitivity to PARP inhibition

Prediction of homologous recombination deficiency identifies colorectal tumors sensitive to PARP inhibition

Homologous Recombination Deficiency Unrelated to Platinum and PARP Inhibitor Response in Cell Line Libraries

The deubiquitylating enzyme USP15 regulates homologous recombination repair and cancer cell response to PARP inhibitors

PALB2 or BARD1 loss confers homologous recombination deficiency and PARP inhibitor sensitivity in prostate cancer

Tumors defective in homologous recombination rely on oxidative metabolism: relevance to treatments with PARP inhibitors

Stellettin B Sensitizes Glioblastoma to DNA‐Damaging Treatments by Suppressing PI3K‐Mediated Homologous Recombination Repair

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CBL0137 impairs homologous recombination repair and sensitizes high-grade serous ovarian carcinoma to PARP inhibitors

Loss of NFBD1/MDC1 disrupts homologous recombination repair and sensitizes nasopharyngeal carcinoma cells to PARP inhibitors

PLK1 overexpression suppresses homologous recombination and confers cellular sensitivity to PARP inhibition

Prediction of homologous recombination deficiency identifies colorectal tumors sensitive to PARP inhibition

Homologous Recombination Deficiency Unrelated to Platinum and PARP Inhibitor Response in Cell Line Libraries

The deubiquitylating enzyme USP15 regulates homologous recombination repair and cancer cell response to PARP inhibitors

PALB2 or BARD1 loss confers homologous recombination deficiency and PARP inhibitor sensitivity in prostate cancer

Tumors defective in homologous recombination rely on oxidative metabolism: relevance to treatments with PARP inhibitors

Stellettin B Sensitizes Glioblastoma to DNA‐Damaging Treatments by Suppressing PI3K‐Mediated Homologous Recombination Repair

Correlation of homologous recombination deficiency induced mutational signatures with sensitivity to PARP inhibitors and cytotoxic agents

Structural basis of homologous recombination

Evaluation of different methods to assess homologous recombination status and sensitivity to PARP inhibitors in ovarian cancer

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Suppression of Homologous Recombination by insulin-like growth factor-1 inhibition sensitizes cancer cells to PARP inhibitors

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Other Objects (12)

CBL0137 impairs homologous recombination repair and sensitizes high-grade serous ovarian carcinoma to PARP inhibitors

Loss of NFBD1/MDC1 disrupts homologous recombination repair and sensitizes nasopharyngeal carcinoma cells to PARP inhibitors

PLK1 overexpression suppresses homologous recombination and confers cellular sensitivity to PARP inhibition

Prediction of homologous recombination deficiency identifies colorectal tumors sensitive to PARP inhibition

Homologous Recombination Deficiency Unrelated to Platinum and PARP Inhibitor Response in Cell Line Libraries

The deubiquitylating enzyme USP15 regulates homologous recombination repair and cancer cell response to PARP inhibitors

PALB2 or BARD1 loss confers homologous recombination deficiency and PARP inhibitor sensitivity in prostate cancer

Tumors defective in homologous recombination rely on oxidative metabolism: relevance to treatments with PARP inhibitors

Stellettin B Sensitizes Glioblastoma to DNA‐Damaging Treatments by Suppressing PI3K‐Mediated Homologous Recombination Repair

Correlation of homologous recombination deficiency induced mutational signatures with sensitivity to PARP inhibitors and cytotoxic agents

Structural basis of homologous recombination

Evaluation of different methods to assess homologous recombination status and sensitivity to PARP inhibitors in ovarian cancer

CBL0137 impairs homologous recombination repair and sensitizes high-grade serous ovarian carcinoma to PARP inhibitors

Loss of NFBD1/MDC1 disrupts homologous recombination repair and sensitizes nasopharyngeal carcinoma cells to PARP inhibitors

PLK1 overexpression suppresses homologous recombination and confers cellular sensitivity to PARP inhibition

Prediction of homologous recombination deficiency identifies colorectal tumors sensitive to PARP inhibition

Homologous Recombination Deficiency Unrelated to Platinum and PARP Inhibitor Response in Cell Line Libraries

The deubiquitylating enzyme USP15 regulates homologous recombination repair and cancer cell response to PARP inhibitors

PALB2 or BARD1 loss confers homologous recombination deficiency and PARP inhibitor sensitivity in prostate cancer

Tumors defective in homologous recombination rely on oxidative metabolism: relevance to treatments with PARP inhibitors

Stellettin B Sensitizes Glioblastoma to DNA‐Damaging Treatments by Suppressing PI3K‐Mediated Homologous Recombination Repair

Correlation of homologous recombination deficiency induced mutational signatures with sensitivity to PARP inhibitors and cytotoxic agents

Structural basis of homologous recombination

Evaluation of different methods to assess homologous recombination status and sensitivity to PARP inhibitors in ovarian cancer

CBL0137 impairs homologous recombination repair and sensitizes high-grade serous ovarian carcinoma to PARP inhibitors

Loss of NFBD1/MDC1 disrupts homologous recombination repair and sensitizes nasopharyngeal carcinoma cells to PARP inhibitors

PLK1 overexpression suppresses homologous recombination and confers cellular sensitivity to PARP inhibition

Prediction of homologous recombination deficiency identifies colorectal tumors sensitive to PARP inhibition

Homologous Recombination Deficiency Unrelated to Platinum and PARP Inhibitor Response in Cell Line Libraries

The deubiquitylating enzyme USP15 regulates homologous recombination repair and cancer cell response to PARP inhibitors

PALB2 or BARD1 loss confers homologous recombination deficiency and PARP inhibitor sensitivity in prostate cancer

Tumors defective in homologous recombination rely on oxidative metabolism: relevance to treatments with PARP inhibitors

Stellettin B Sensitizes Glioblastoma to DNA‐Damaging Treatments by Suppressing PI3K‐Mediated Homologous Recombination Repair

Correlation of homologous recombination deficiency induced mutational signatures with sensitivity to PARP inhibitors and cytotoxic agents

Structural basis of homologous recombination

Evaluation of different methods to assess homologous recombination status and sensitivity to PARP inhibitors in ovarian cancer

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