PDK4 rescues high-glucose-induced senescent fibroblasts and promotes diabetic wound healing through enhancing glycolysis and regulating YAP and JNK pathway
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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PDK4 rescues high-glucose-induced senescent fibroblasts and promotes diabetic wound healing through enhancing glycolysis and regulating YAP and JNK pathway ; volume:9 ; number:1 ; day:25 ; month:11 ; year:2023 ; pages:1-15 ; date:12.2023
Cell death discovery ; 9, Heft 1 (25.11.2023), 1-15, 12.2023
- Creator
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Ma, Zhouji
Ding, Youjun
Ding, Xiaofeng
Mou, Haining
Mo, Ran
Tan, Qian
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41420-023-01725-2
- URN
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urn:nbn:de:101:1-2024021310414469846751
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:21 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Ma, Zhouji
- Ding, Youjun
- Ding, Xiaofeng
- Mou, Haining
- Mo, Ran
- Tan, Qian
- SpringerLink (Online service)
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Mutual repression between JNK/AP-1 and JAK/STAT stratifies senescent and proliferative cell behaviors during tissue regeneration
p21 maintains senescent cell viability under persistent DNA damage response by restraining JNK and caspase signaling
YAP/TAZ activation mediates PQ-induced lung fibrosis by sustaining senescent pulmonary epithelial cells
Senescent cells promote breast cancer cells motility by secreting GM-CSF and bFGF that activate the JNK signaling pathway
The diseased kidney: aging and senescent immunology
Blockade of Crk eliminates Yki/YAP-activated tumors via JNK-mediated apoptosis in Drosophila
SKP2- and OTUD1-regulated non-proteolytic ubiquitination of YAP promotes YAP nuclear localization and activity
Distribution and impact of p16INK4A+ senescent cells in elderly tissues: a focus on senescent immune cell and epithelial dysfunction
Circulating Angiogenic and Senescent T Lymphocytes in Ageing and Frailty
Senescent glia link mitochondrial dysfunction and lipid accumulation
Mst1 overexpression combined with Yap knockdown augments thyroid carcinoma apoptosis via promoting MIEF1-related mitochondrial fission and activating the JNK pathway
Curcumin derivative WZ35 inhibits tumor cell growth via ROS-YAP-JNK signaling pathway in breast cancer
Mutual repression between JNK/AP-1 and JAK/STAT stratifies senescent and proliferative cell behaviors during tissue regeneration
p21 maintains senescent cell viability under persistent DNA damage response by restraining JNK and caspase signaling
YAP/TAZ activation mediates PQ-induced lung fibrosis by sustaining senescent pulmonary epithelial cells
Senescent cells promote breast cancer cells motility by secreting GM-CSF and bFGF that activate the JNK signaling pathway
The diseased kidney: aging and senescent immunology
Blockade of Crk eliminates Yki/YAP-activated tumors via JNK-mediated apoptosis in Drosophila
SKP2- and OTUD1-regulated non-proteolytic ubiquitination of YAP promotes YAP nuclear localization and activity
Distribution and impact of p16INK4A+ senescent cells in elderly tissues: a focus on senescent immune cell and epithelial dysfunction
Circulating Angiogenic and Senescent T Lymphocytes in Ageing and Frailty
Senescent glia link mitochondrial dysfunction and lipid accumulation
Mst1 overexpression combined with Yap knockdown augments thyroid carcinoma apoptosis via promoting MIEF1-related mitochondrial fission and activating the JNK pathway