- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1742-2094
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Amelioration of autoimmune neuroinflammation by the fusion molecule Fn14·TRAIL ; volume:10 ; number:1 ; day:9 ; month:3 ; year:2013 ; pages:1-11 ; date:12.2013
Journal of neuroinflammation ; 10, Heft 1 (9.3.2013), 1-11, 12.2013
- Creator
-
Prinz-Hadad, Hodaya
- Contributor
-
Mizrachi, Tehila
Irony-Tur-Sinai, Michal
Prigozhina, Tatyana B.
Aronin, Alexandra
Brenner, Talma
Dranitzki-Elhalel, Michal
SpringerLink (Online service)
- DOI
-
10.1186/1742-2094-10-36
- URN
-
urn:nbn:de:1111-2016032334686
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:44 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Prinz-Hadad, Hodaya
- Mizrachi, Tehila
- Irony-Tur-Sinai, Michal
- Prigozhina, Tatyana B.
- Aronin, Alexandra
- Brenner, Talma
- Dranitzki-Elhalel, Michal
- SpringerLink (Online service)
Other Objects (12)
Cytomegalovirus infection exacerbates autoimmune mediated neuroinflammation
Adenosine 2a receptor signaling in autoimmune neuroinflammation
Regulation of autoimmune‐mediated neuroinflammation by endothelial cells
Regulation of autoimmune neuroinflammation by Hv1 proton channels
Dual roles of the adenosine A2a receptor in autoimmune neuroinflammation
Chi3l3 induces oligodendrogenesis in an experimental model of autoimmune neuroinflammation
Effects of Glatiramer acetate in a spontaneous model of autoimmune neuroinflammation
Lowering blood cholesterol does not affect neuroinflammation in experimental autoimmune encephalomyelitis
Exacerbating effects of single-dose acute ethanol exposure on neuroinflammation and amelioration by GPR110 (ADGRF1) activation
Amelioration of experimental autoimmune encephalomyelitis by in vivo reprogramming of macrophages using pro-resolving factors
Dietary galactose exacerbates autoimmune neuroinflammation via advanced glycation end product-mediated neurodegeneration
A small-molecule TLR4 antagonist reduced neuroinflammation in female E4FAD mice
Cytomegalovirus infection exacerbates autoimmune mediated neuroinflammation
Adenosine 2a receptor signaling in autoimmune neuroinflammation
Regulation of autoimmune‐mediated neuroinflammation by endothelial cells
Regulation of autoimmune neuroinflammation by Hv1 proton channels
Dual roles of the adenosine A2a receptor in autoimmune neuroinflammation
Chi3l3 induces oligodendrogenesis in an experimental model of autoimmune neuroinflammation
Effects of Glatiramer acetate in a spontaneous model of autoimmune neuroinflammation
Lowering blood cholesterol does not affect neuroinflammation in experimental autoimmune encephalomyelitis
Exacerbating effects of single-dose acute ethanol exposure on neuroinflammation and amelioration by GPR110 (ADGRF1) activation
Amelioration of experimental autoimmune encephalomyelitis by in vivo reprogramming of macrophages using pro-resolving factors
Dietary galactose exacerbates autoimmune neuroinflammation via advanced glycation end product-mediated neurodegeneration
A small-molecule TLR4 antagonist reduced neuroinflammation in female E4FAD mice
Cytomegalovirus infection exacerbates autoimmune mediated neuroinflammation
Adenosine 2a receptor signaling in autoimmune neuroinflammation
Regulation of autoimmune‐mediated neuroinflammation by endothelial cells
Regulation of autoimmune neuroinflammation by Hv1 proton channels
Dual roles of the adenosine A2a receptor in autoimmune neuroinflammation
Chi3l3 induces oligodendrogenesis in an experimental model of autoimmune neuroinflammation
Effects of Glatiramer acetate in a spontaneous model of autoimmune neuroinflammation
Lowering blood cholesterol does not affect neuroinflammation in experimental autoimmune encephalomyelitis
Exacerbating effects of single-dose acute ethanol exposure on neuroinflammation and amelioration by GPR110 (ADGRF1) activation
Amelioration of experimental autoimmune encephalomyelitis by in vivo reprogramming of macrophages using pro-resolving factors
Dietary galactose exacerbates autoimmune neuroinflammation via advanced glycation end product-mediated neurodegeneration