“Dual Disease” TgAD/GSS mice exhibit enhanced Alzheimer’s disease pathology and reveal PrPC-dependent secretion of Aβ
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2045-2322
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
“Dual Disease” TgAD/GSS mice exhibit enhanced Alzheimer’s disease pathology and reveal PrPC-dependent secretion of Aβ ; volume:9 ; number:1 ; day:12 ; month:6 ; year:2019 ; pages:1-18 ; date:12.2019
Scientific reports ; 9, Heft 1 (12.6.2019), 1-18, 12.2019
- Creator
-
Qin, Kefeng
Zhao, Lili
Gregory, Crystal
Solanki, Ani
Mastrianni, James A.
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1038/s41598-019-44317-w
- URN
-
urn:nbn:de:101:1-2019101812090476566818
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:39 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Qin, Kefeng
- Zhao, Lili
- Gregory, Crystal
- Solanki, Ani
- Mastrianni, James A.
- SpringerLink (Online service)
Other Objects (12)
Alzheimer’s Disease
Spatial memory deficiency early in 6xTg Alzheimer’s disease mouse model
Alzheimer's disease
Human Alzheimer’s disease reactive astrocytes exhibit a loss of homeostastic gene expression
Anti-HBs conjugated superparamagnetic iron oxide nanoparticles (SPION) to visualize and target HBV infection in vitro and in vivo
Bonding in M(NHBMe)2 and M[Mn(CO)5]2 complexes (M=Zn, Cd, Hg; NHBMe=(HCNMe)2B): divalent group 12 metals with zero oxidation state
Genetic Features of Young and Aged Animals After Peripheral Nerve Injury: Implications for Diminished Regeneration Capacity
Disease-modifying treatments in Alzheimer’s disease
Alzheimer’s disease as an inflammatory disease
Cerebrovascular disease in ageing and Alzheimer’s disease
Endocytosis and Alzheimer’s disease
Biomarkers in Alzheimer’s disease
Alzheimer’s Disease
Spatial memory deficiency early in 6xTg Alzheimer’s disease mouse model
Alzheimer's disease
Human Alzheimer’s disease reactive astrocytes exhibit a loss of homeostastic gene expression
Anti-HBs conjugated superparamagnetic iron oxide nanoparticles (SPION) to visualize and target HBV infection in vitro and in vivo
Bonding in M(NHBMe)2 and M[Mn(CO)5]2 complexes (M=Zn, Cd, Hg; NHBMe=(HCNMe)2B): divalent group 12 metals with zero oxidation state
Genetic Features of Young and Aged Animals After Peripheral Nerve Injury: Implications for Diminished Regeneration Capacity
Disease-modifying treatments in Alzheimer’s disease
Alzheimer’s disease as an inflammatory disease
Cerebrovascular disease in ageing and Alzheimer’s disease
Endocytosis and Alzheimer’s disease
Biomarkers in Alzheimer’s disease
Alzheimer’s Disease
Spatial memory deficiency early in 6xTg Alzheimer’s disease mouse model
Alzheimer's disease
Human Alzheimer’s disease reactive astrocytes exhibit a loss of homeostastic gene expression
Anti-HBs conjugated superparamagnetic iron oxide nanoparticles (SPION) to visualize and target HBV infection in vitro and in vivo
Bonding in M(NHBMe)2 and M[Mn(CO)5]2 complexes (M=Zn, Cd, Hg; NHBMe=(HCNMe)2B): divalent group 12 metals with zero oxidation state
Genetic Features of Young and Aged Animals After Peripheral Nerve Injury: Implications for Diminished Regeneration Capacity
Disease-modifying treatments in Alzheimer’s disease
Alzheimer’s disease as an inflammatory disease
Cerebrovascular disease in ageing and Alzheimer’s disease
Endocytosis and Alzheimer’s disease