A dual role for glucocorticoid-induced leucine zipper in glucocorticoid function: tumor growth promotion or suppression?
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2041-4889
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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A dual role for glucocorticoid-induced leucine zipper in glucocorticoid function: tumor growth promotion or suppression? ; volume:9 ; number:5 ; day:26 ; month:4 ; year:2018 ; pages:1-12 ; date:5.2018
Cell death & disease ; 9, Heft 5 (26.4.2018), 1-12, 5.2018
- Creator
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Ayroldi, Emira
- Contributor
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Cannarile, Lorenza
Delfino, Domenico V.
Riccardi, Carlo
SpringerLink (Online service)
- DOI
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10.1038/s41419-018-0558-1
- URN
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urn:nbn:de:101:1-2018082200510466639176
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:25 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Ayroldi, Emira
- Cannarile, Lorenza
- Delfino, Domenico V.
- Riccardi, Carlo
- SpringerLink (Online service)
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Regulation of glucocorticoid-induced leucine zipper (GILZ) in vascular inflammation
Long glucocorticoid-induced leucine zipper regulates human thyroid cancer cell proliferation
Glucocorticoid-induced leucine zipper (GILZ) is involved in glucocorticoid-induced and mineralocorticoid-induced leptin production by osteoarthritis synovial fibroblasts
Regulation des Glucocorticoid-induced leucine zipper (GILZ) in Entzündungsprozessen : Mechanismen und funktionelle Bedeutung
Influence of the glucocorticoid-induced leucine zipper (GILZ) on macrophage-associated inflammation and statin-mediated effects
Identification of glucocorticoid-induced leucine zipper as a key regulator of tumor cell proliferation in epithelial ovarian cancer
Regulation of toll-like receptor expression by glucocorticoid-induced leucine zipper and p38 MAPK in human endothelial cells
Neutrophil expression of glucocorticoid-induced leucine zipper (GILZ) anti-inflammatory protein is associated with acute respiratory distress syndrome severity
Glucocorticoid receptor in stromal cells is essential for glucocorticoid-mediated suppression of inflammation in arthritis
The association between the rs3747406 polymorphism in the glucocorticoid-induced leucine zipper gene and sepsis survivals depends on the SOFA score
Investigations on the role of the glucocorticoid-induced leucine zipper in macrophage activation with a focus on sodium-mediated inflammation and trained immunity
Glucocorticoid receptor GRβ regulates glucocorticoid-induced ocular hypertension in mice
Regulation of glucocorticoid-induced leucine zipper (GILZ) in vascular inflammation
Long glucocorticoid-induced leucine zipper regulates human thyroid cancer cell proliferation
Glucocorticoid-induced leucine zipper (GILZ) is involved in glucocorticoid-induced and mineralocorticoid-induced leptin production by osteoarthritis synovial fibroblasts
Regulation des Glucocorticoid-induced leucine zipper (GILZ) in Entzündungsprozessen : Mechanismen und funktionelle Bedeutung
Influence of the glucocorticoid-induced leucine zipper (GILZ) on macrophage-associated inflammation and statin-mediated effects
Identification of glucocorticoid-induced leucine zipper as a key regulator of tumor cell proliferation in epithelial ovarian cancer
Regulation of toll-like receptor expression by glucocorticoid-induced leucine zipper and p38 MAPK in human endothelial cells
Neutrophil expression of glucocorticoid-induced leucine zipper (GILZ) anti-inflammatory protein is associated with acute respiratory distress syndrome severity
Glucocorticoid receptor in stromal cells is essential for glucocorticoid-mediated suppression of inflammation in arthritis
The association between the rs3747406 polymorphism in the glucocorticoid-induced leucine zipper gene and sepsis survivals depends on the SOFA score
Investigations on the role of the glucocorticoid-induced leucine zipper in macrophage activation with a focus on sodium-mediated inflammation and trained immunity