Selected microRNAs Increase Synaptic Resilience to the Damaging Binding of the Alzheimer’s Disease Amyloid Beta Oligomers
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1559-1182
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Selected microRNAs Increase Synaptic Resilience to the Damaging Binding of the Alzheimer’s Disease Amyloid Beta Oligomers ; volume:57 ; number:5 ; day:29 ; month:1 ; year:2020 ; pages:2232-2243 ; date:5.2020
Molecular neurobiology ; 57, Heft 5 (29.1.2020), 2232-2243, 5.2020
- Creator
-
Zolochevska, Olga
Taglialatela, Giulio
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1007/s12035-020-01868-8
- URN
-
urn:nbn:de:101:1-2020063022333183343778
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:54 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Zolochevska, Olga
- Taglialatela, Giulio
- SpringerLink (Online service)
Other Objects (12)
Altered microRNAs related to synaptic function as potential plasma biomarkers for Alzheimer’s disease
APOE4/4 is linked to damaging lipid droplets in Alzheimer’s disease microglia
Role of BACE1 in Alzheimer’s synaptic function
Alzheimer’s disease: presence and role of microRNAs
Alzheimer’s disease pathology propagation by exosomes containing toxic amyloid-beta oligomers
Cellular Receptors of Amyloid β Oligomers (AβOs) in Alzheimer’s Disease
Oligomeric Aβ-induced synaptic dysfunction in Alzheimer’s disease
MicroRNAs circulate around Alzheimer's disease
Synaptosome microRNAs regulate synapse functions in Alzheimer’s disease
Alzheimer’s disease: Amyloid oligomers, therapeutic agents, their target and mode of action
Prion protein oligomers cause neuronal cytoskeletal damage in rapidly progressive Alzheimer’s disease
Aβ oligomers trigger necroptosis-mediated neurodegeneration via microglia activation in Alzheimer’s disease
Altered microRNAs related to synaptic function as potential plasma biomarkers for Alzheimer’s disease
APOE4/4 is linked to damaging lipid droplets in Alzheimer’s disease microglia
Role of BACE1 in Alzheimer’s synaptic function
Alzheimer’s disease: presence and role of microRNAs
Alzheimer’s disease pathology propagation by exosomes containing toxic amyloid-beta oligomers
Cellular Receptors of Amyloid β Oligomers (AβOs) in Alzheimer’s Disease
Oligomeric Aβ-induced synaptic dysfunction in Alzheimer’s disease
MicroRNAs circulate around Alzheimer's disease
Synaptosome microRNAs regulate synapse functions in Alzheimer’s disease
Alzheimer’s disease: Amyloid oligomers, therapeutic agents, their target and mode of action
Prion protein oligomers cause neuronal cytoskeletal damage in rapidly progressive Alzheimer’s disease
Aβ oligomers trigger necroptosis-mediated neurodegeneration via microglia activation in Alzheimer’s disease
Altered microRNAs related to synaptic function as potential plasma biomarkers for Alzheimer’s disease
APOE4/4 is linked to damaging lipid droplets in Alzheimer’s disease microglia
Role of BACE1 in Alzheimer’s synaptic function
Alzheimer’s disease: presence and role of microRNAs
Alzheimer’s disease pathology propagation by exosomes containing toxic amyloid-beta oligomers
Cellular Receptors of Amyloid β Oligomers (AβOs) in Alzheimer’s Disease
Oligomeric Aβ-induced synaptic dysfunction in Alzheimer’s disease
MicroRNAs circulate around Alzheimer's disease
Synaptosome microRNAs regulate synapse functions in Alzheimer’s disease
Alzheimer’s disease: Amyloid oligomers, therapeutic agents, their target and mode of action
Prion protein oligomers cause neuronal cytoskeletal damage in rapidly progressive Alzheimer’s disease