A SGLT2 inhibitor dapagliflozin suppresses prolonged ventricular-repolarization through augmentation of mitochondrial function in insulin-resistant metabolic syndrome rats
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1475-2840
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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A SGLT2 inhibitor dapagliflozin suppresses prolonged ventricular-repolarization through augmentation of mitochondrial function in insulin-resistant metabolic syndrome rats ; volume:17 ; number:1 ; day:17 ; month:11 ; year:2018 ; pages:1-17 ; date:12.2018
Cardiovascular diabetology ; 17, Heft 1 (17.11.2018), 1-17, 12.2018
- Creator
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Durak, Aysegul
Olgar, Yusuf
Degirmenci, Sinan
Akkus, Erman
Tuncay, Erkan
Turan, Belma
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s12933-018-0790-0
- URN
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urn:nbn:de:101:1-2019010119354329160103
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:44 AM CEST
Data provider
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Associated
- Durak, Aysegul
- Olgar, Yusuf
- Degirmenci, Sinan
- Akkus, Erman
- Tuncay, Erkan
- Turan, Belma
- SpringerLink (Online service)
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Evaluation of the effect of dapagliflozin on cardiac repolarization: a thorough QT/QTc study
Acute antiarrhythmic effects of SGLT2 inhibitors–dapagliflozin lowers the excitability of atrial cardiomyocytes
Highly selective SGLT2 inhibitor dapagliflozin reduces seizure activity in pentylenetetrazol-induced murine model of epilepsy
Dapagliflozin targets SGLT2/SIRT1 signaling to attenuate the osteogenic transdifferentiation of vascular smooth muscle cells
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