Hochschulschrift
Mitochondrial reactive oxygen species : molecular mechanisms, biomarkers and their role in permeability transition
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Dimensions
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30 cm
- Extent
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III, 236 S.
- Language
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Englisch
- Notes
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Ill., graph. Darst.
Frankfurt (Main), Univ., Diss., 2013
- Keyword
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Mitochondrium
Sauerstoff
Bioenergetik
Biochemische Reaktion
Biophysikalische Chemie
Energiestoffwechsel
- Creator
- Table of contents
- Rights
-
Bei diesem Objekt liegt nur das Inhaltsverzeichnis digital vor. Der Zugriff darauf ist unbeschränkt möglich.
- Last update
-
11.06.2025, 2:15 PM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Object type
- Hochschulschrift
Associated
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Progress in understanding the molecular oxygen paradox – function of mitochondrial reactive oxygen species in cell signaling
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Targeting mitochondrial reactive oxygen species as novel therapy for inflammatory diseases and cancers
Palmitic Acid-Induced Podocyte Apoptosis via the Reactive Oxygen Species-Dependent Mitochondrial Pathway
Subclinical dose irradiation triggers human breast cancer migration via mitochondrial reactive oxygen species
Alterations in mitochondrial respiration and reactive oxygen species in patients poisoned with carbon monoxide treated with hyperbaric oxygen
Iron oxide nanoparticles induce human microvascular endothelial cell permeability through reactive oxygen species production and microtubule remodeling
Mitochondrial DNA abnormalities provide mechanistic insight and predict reactive oxygen species-stimulating drug efficacy
Double-activation of mitochondrial permeability transition pore opening via calcium overload and reactive oxygen species for cancer therapy
Anticancer Aminoferrocene Derivatives Inducing Production of Mitochondrial Reactive Oxygen Species
Molecular dynamic simulations of oxidized skin lipid bilayer and permeability of reactive oxygen species
Respiratory uncoupling by increased H+ or K+ flux is beneficial for heart mitochondrial turnover of reactive oxygen species but not for permeability transition
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Subclinical dose irradiation triggers human breast cancer migration via mitochondrial reactive oxygen species
Alterations in mitochondrial respiration and reactive oxygen species in patients poisoned with carbon monoxide treated with hyperbaric oxygen
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