Nuclear lamina dysfunction triggers a germline stem cell checkpoint

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2041-1723

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Nuclear lamina dysfunction triggers a germline stem cell checkpoint ; volume:9 ; number:1 ; day:27 ; month:9 ; year:2018 ; pages:1-13 ; date:12.2018
Nature Communications ; 9, Heft 1 (27.9.2018), 1-13, 12.2018

Classification: Biowissenschaften, Biologie

Creator: Barton, Lacy J.

Contributor: Duan, Tingting
Ke, Wenfan
Luttinger, Amy
Lovander, Kaylee E.
Soshnev, Alexey A.
Geyer, Pamela K.
SpringerLink (Online service)

DOI: 10.1038/s41467-018-06277-z

URN: urn:nbn:de:101:1-2018110600052518176714

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 11:03 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.

Show original at data provider

Associated

Barton, Lacy J.
Duan, Tingting
Ke, Wenfan
Luttinger, Amy
Lovander, Kaylee E.
Soshnev, Alexey A.
Geyer, Pamela K.
SpringerLink (Online service)

Other Objects (12)

Germline genetic variation and predicting immune checkpoint inhibitor induced toxicity

Germline HAVCR2/TIM-3 Checkpoint Inhibitor Receptor Deficiency in Recurrent Autoinflammatory Myocarditis

Illustration

[Lamina XVI - Lamina XVII]

A germline-to-soma signal triggers an age-related decline of mitochondrial stress response

Illustration

Lámina I. [y] Lámina II.

Mitochondrial HSF1 triggers mitochondrial dysfunction and neurodegeneration in Huntington's disease

Abschnitt

Lámina

Cellular Alterations in Immune Checkpoint Inhibitor Therapy-Related Cardiac Dysfunction

Dysfunction of Lamin B and Physiological Aging Cause Telomere Instability in Drosophila Germline

Compound heterozygous MSH3 germline variants and associated tumor somatic DNA mismatch repair dysfunction

Illustration

Lámina XIX.

Illustration

Lámina XVIII.

Germline genetic variation and predicting immune checkpoint inhibitor induced toxicity

Germline HAVCR2/TIM-3 Checkpoint Inhibitor Receptor Deficiency in Recurrent Autoinflammatory Myocarditis

Illustration

[Lamina XVI - Lamina XVII]

A germline-to-soma signal triggers an age-related decline of mitochondrial stress response

Illustration

Lámina I. [y] Lámina II.

Mitochondrial HSF1 triggers mitochondrial dysfunction and neurodegeneration in Huntington's disease

Abschnitt

Lámina

Cellular Alterations in Immune Checkpoint Inhibitor Therapy-Related Cardiac Dysfunction

Dysfunction of Lamin B and Physiological Aging Cause Telomere Instability in Drosophila Germline

Compound heterozygous MSH3 germline variants and associated tumor somatic DNA mismatch repair dysfunction

Illustration

Lámina XIX.

Illustration

Lámina XVIII.

Germline genetic variation and predicting immune checkpoint inhibitor induced toxicity

Germline HAVCR2/TIM-3 Checkpoint Inhibitor Receptor Deficiency in Recurrent Autoinflammatory Myocarditis

Illustration

[Lamina XVI - Lamina XVII]

A germline-to-soma signal triggers an age-related decline of mitochondrial stress response

Illustration

Lámina I. [y] Lámina II.

Mitochondrial HSF1 triggers mitochondrial dysfunction and neurodegeneration in Huntington's disease

Abschnitt

Lámina

Cellular Alterations in Immune Checkpoint Inhibitor Therapy-Related Cardiac Dysfunction

Dysfunction of Lamin B and Physiological Aging Cause Telomere Instability in Drosophila Germline

Compound heterozygous MSH3 germline variants and associated tumor somatic DNA mismatch repair dysfunction

Illustration

Lámina XIX.

Illustration

Lámina XVIII.

Cultural heritage institutions wishing to register will find more information here.

Fields marked * need to be filled in.

Username*

Please enter your username

Email*

Please enter your email address

Please do not fill this field

First name

Last name

Password*

Please enter your password

Confirm password*

Please enter the same password

I have read the terms of use and the privacy policy for the collection of personal data and accept them. *

This field is required.

I would like to subscribe to the newsletter of the Deutsche Digitale Bibliothek. See newsletter subscription info.

Account created

Your "My DDB" account has been successfully created. Before you can log in to your account, you must click the confirmation link in the message we just sent to the email address you provided.

Nuclear lamina dysfunction triggers a germline stem cell checkpoint

Object Details

References and Relationships

Classification and Topics

Contributors, Places and Time

Further information

Data provider

Associated

Other Objects (12)

Germline genetic variation and predicting immune checkpoint inhibitor induced toxicity

Germline HAVCR2/TIM-3 Checkpoint Inhibitor Receptor Deficiency in Recurrent Autoinflammatory Myocarditis

[Lamina XVI - Lamina XVII]

A germline-to-soma signal triggers an age-related decline of mitochondrial stress response

Lámina I. [y] Lámina II.

Mitochondrial HSF1 triggers mitochondrial dysfunction and neurodegeneration in Huntington's disease

Lámina

Cellular Alterations in Immune Checkpoint Inhibitor Therapy-Related Cardiac Dysfunction

Dysfunction of Lamin B and Physiological Aging Cause Telomere Instability in Drosophila Germline

Compound heterozygous MSH3 germline variants and associated tumor somatic DNA mismatch repair dysfunction

Lámina XIX.

Lámina XVIII.

Germline genetic variation and predicting immune checkpoint inhibitor induced toxicity

Germline HAVCR2/TIM-3 Checkpoint Inhibitor Receptor Deficiency in Recurrent Autoinflammatory Myocarditis

[Lamina XVI - Lamina XVII]

A germline-to-soma signal triggers an age-related decline of mitochondrial stress response

Lámina I. [y] Lámina II.

Mitochondrial HSF1 triggers mitochondrial dysfunction and neurodegeneration in Huntington's disease

Lámina

Cellular Alterations in Immune Checkpoint Inhibitor Therapy-Related Cardiac Dysfunction

Dysfunction of Lamin B and Physiological Aging Cause Telomere Instability in Drosophila Germline

Compound heterozygous MSH3 germline variants and associated tumor somatic DNA mismatch repair dysfunction

Lámina XIX.

Lámina XVIII.

Germline genetic variation and predicting immune checkpoint inhibitor induced toxicity

Germline HAVCR2/TIM-3 Checkpoint Inhibitor Receptor Deficiency in Recurrent Autoinflammatory Myocarditis

[Lamina XVI - Lamina XVII]

A germline-to-soma signal triggers an age-related decline of mitochondrial stress response

Lámina I. [y] Lámina II.

Mitochondrial HSF1 triggers mitochondrial dysfunction and neurodegeneration in Huntington's disease

Lámina

Cellular Alterations in Immune Checkpoint Inhibitor Therapy-Related Cardiac Dysfunction

Dysfunction of Lamin B and Physiological Aging Cause Telomere Instability in Drosophila Germline

Compound heterozygous MSH3 germline variants and associated tumor somatic DNA mismatch repair dysfunction

Lámina XIX.

Lámina XVIII.

Related objects

Reset password