Intestinal Protein Characterisation of SARS-CoV-2 Entry Molecules ACE2 and TMPRSS2 in Inflammatory Bowel Disease (IBD) and Fatal COVID-19 Infection
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1573-2576
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Intestinal Protein Characterisation of SARS-CoV-2 Entry Molecules ACE2 and TMPRSS2 in Inflammatory Bowel Disease (IBD) and Fatal COVID-19 Infection ; day:25 ; month:10 ; year:2021 ; pages:1-6
Inflammation ; (25.10.2021), 1-6
- Creator
-
McAllister, Milly J.
Kirkwood, Kathryn
Chuah, Shaun C.
Thompson, Emily J.
Cartwright, Jennifer A.
Russell, Clark D.
Dorward, David A.
Lucas, Christopher D.
Ho, Gwo-tzer
- Contributor
-
SpringerLink (Online service)
- DOI
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10.1007/s10753-021-01567-z
- URN
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urn:nbn:de:101:1-2022010521160221591408
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:23 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- McAllister, Milly J.
- Kirkwood, Kathryn
- Chuah, Shaun C.
- Thompson, Emily J.
- Cartwright, Jennifer A.
- Russell, Clark D.
- Dorward, David A.
- Lucas, Christopher D.
- Ho, Gwo-tzer
- SpringerLink (Online service)
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Coronaviruses Use ACE2 Monomers as Entry‐Receptors
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Analyse von sACE2 und sTMPRSS2 bei Kindern und Erwachsenen mit und ohne COVID-19
Association of ACE2 and TMPRSS2 towards COVID-19 susceptibility
Sputum ACE2, TMPRSS2 and FURIN gene expression in severe neutrophilic asthma
MicroRNAs modulate SARS-CoV-2 infection of primary human hepatocytes by regulating the entry factors ACE2 and TMPRSS2
No evidence that ACE2 or TMPRSS2 drive population disparity in COVID risks
Coronaviruses Use ACE2 Monomers as Entry‐Receptors
Coronaviruses Use ACE2 Monomers as Entry‐Receptors
Age and gender differences in ACE2 and TMPRSS2 expressions in oral epithelial cells
Correction to: Sputum ACE2, TMPRSS2 and FURIN gene expression in severe neutrophilic asthma
Nanoengineered Red Blood Cells Loaded with TMPRSS2 and Cathepsin L Inhibitors Block SARS‐CoV‐2 Pseudovirus Entry into Lung ACE2 + Cells
New insights into genetic susceptibility of COVID-19: an ACE2 and TMPRSS2 polymorphism analysis
Analyzing the role of ACE2, AR, MX1 and TMPRSS2 genetic markers for COVID-19 severity
Analyse von sACE2 und sTMPRSS2 bei Kindern und Erwachsenen mit und ohne COVID-19
Association of ACE2 and TMPRSS2 towards COVID-19 susceptibility
Sputum ACE2, TMPRSS2 and FURIN gene expression in severe neutrophilic asthma
MicroRNAs modulate SARS-CoV-2 infection of primary human hepatocytes by regulating the entry factors ACE2 and TMPRSS2
No evidence that ACE2 or TMPRSS2 drive population disparity in COVID risks
Coronaviruses Use ACE2 Monomers as Entry‐Receptors
Coronaviruses Use ACE2 Monomers as Entry‐Receptors
Age and gender differences in ACE2 and TMPRSS2 expressions in oral epithelial cells
Correction to: Sputum ACE2, TMPRSS2 and FURIN gene expression in severe neutrophilic asthma
Nanoengineered Red Blood Cells Loaded with TMPRSS2 and Cathepsin L Inhibitors Block SARS‐CoV‐2 Pseudovirus Entry into Lung ACE2 + Cells
New insights into genetic susceptibility of COVID-19: an ACE2 and TMPRSS2 polymorphism analysis
Analyzing the role of ACE2, AR, MX1 and TMPRSS2 genetic markers for COVID-19 severity