DAD3 targets ACE2 to inhibit the MAPK and NF-κB signalling pathways and protect against LPS-induced inflammation in bovine mammary epithelial cells

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1297-9716

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: DAD3 targets ACE2 to inhibit the MAPK and NF-κB signalling pathways and protect against LPS-induced inflammation in bovine mammary epithelial cells ; volume:53 ; number:1 ; day:8 ; month:12 ; year:2022 ; pages:1-13 ; date:12.2022
Veterinary research ; 53, Heft 1 (8.12.2022), 1-13, 12.2022

Creator: Zhang, Xiangjun
Jia, Fang
Ma, Weiwu
Li, Xueqiang
Zhou, Xuezhang

Contributor: SpringerLink (Online service)

DOI: 10.1186/s13567-022-01122-0

URN: urn:nbn:de:101:1-2023021809094015661303

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 11:01 AM CEST

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Zhang, Xiangjun
Jia, Fang
Ma, Weiwu
Li, Xueqiang
Zhou, Xuezhang
SpringerLink (Online service)

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DAD3 targets ACE2 to inhibit the MAPK and NF-κB signalling pathways and protect against LPS-induced inflammation in bovine mammary epithelial cells

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Other Objects (12)

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Inhibition of EGFR-MAPK signaling reduces ACE2-dependent SARS-CoV-2 infection.

Mammary adipocytes protect triple-negative breast cancer cells from ferroptosis

FXR inhibition may protect from SARS-CoV-2 infection by reducing ACE2

Progranulin inhibits LPS-induced macrophage M1 polarization via NF-кB and MAPK pathways

Role of TLR4-p38 MAPK-Hsp27 signal pathway in LPS-induced pulmonary epithelial hyperpermeability

Microglial p38α MAPK is critical for LPS-induced neuron degeneration, through a mechanism involving TNFα

Anadenanthera colubrina regulated LPS-induced inflammation by suppressing NF-κB and p38-MAPK signaling pathways

P38 MAPK activated ADAM17 mediates ACE2 shedding and promotes cardiac remodeling and heart failure after myocardial infarction

Immunohistochemical study for the expression of leukocyte adhesion molecules, and FGF23 and ACE2 in P. gingivalis LPS-induced diabetic nephropathy

ACE2-EGFR-MAPK signaling contributes to SARS-CoV-2 infection

(+)-JQ1 attenuated LPS-induced microglial inflammation via MAPK/NFκB signaling

Daphnetin may protect from SARS-CoV-2 infection by reducing ACE2

Inhibition of EGFR-MAPK signaling reduces ACE2-dependent SARS-CoV-2 infection.

Mammary adipocytes protect triple-negative breast cancer cells from ferroptosis

FXR inhibition may protect from SARS-CoV-2 infection by reducing ACE2

Progranulin inhibits LPS-induced macrophage M1 polarization via NF-кB and MAPK pathways

Role of TLR4-p38 MAPK-Hsp27 signal pathway in LPS-induced pulmonary epithelial hyperpermeability

Microglial p38α MAPK is critical for LPS-induced neuron degeneration, through a mechanism involving TNFα

Anadenanthera colubrina regulated LPS-induced inflammation by suppressing NF-κB and p38-MAPK signaling pathways

P38 MAPK activated ADAM17 mediates ACE2 shedding and promotes cardiac remodeling and heart failure after myocardial infarction

Immunohistochemical study for the expression of leukocyte adhesion molecules, and FGF23 and ACE2 in P. gingivalis LPS-induced diabetic nephropathy

ACE2-EGFR-MAPK signaling contributes to SARS-CoV-2 infection

(+)-JQ1 attenuated LPS-induced microglial inflammation via MAPK/NFκB signaling

Daphnetin may protect from SARS-CoV-2 infection by reducing ACE2

Inhibition of EGFR-MAPK signaling reduces ACE2-dependent SARS-CoV-2 infection.

Mammary adipocytes protect triple-negative breast cancer cells from ferroptosis

FXR inhibition may protect from SARS-CoV-2 infection by reducing ACE2

Progranulin inhibits LPS-induced macrophage M1 polarization via NF-кB and MAPK pathways

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P38 MAPK activated ADAM17 mediates ACE2 shedding and promotes cardiac remodeling and heart failure after myocardial infarction

Immunohistochemical study for the expression of leukocyte adhesion molecules, and FGF23 and ACE2 in P. gingivalis LPS-induced diabetic nephropathy

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