Release of soluble vascular endothelial growth factor receptor-1 (sFlt-1) during coronary artery bypass surgery
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1749-8090
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Release of soluble vascular endothelial growth factor receptor-1 (sFlt-1) during coronary artery bypass surgery ; volume:2 ; number:1 ; day:21 ; month:9 ; year:2007 ; pages:1-7 ; date:12.2007
Journal of cardiothoracic surgery ; 2, Heft 1 (21.9.2007), 1-7, 12.2007
- Creator
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Denizot, Yves
- Contributor
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Leguyader, Alexandre
Cornu, Elisabeth
Laskar, Marc
Orsel, Isabelle
Vincent, Christelle
Nathan, Nathalie
SpringerLink (Online service)
- DOI
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10.1186/1749-8090-2-38
- URN
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urn:nbn:de:1111-2016070928075
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:54 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Denizot, Yves
- Leguyader, Alexandre
- Cornu, Elisabeth
- Laskar, Marc
- Orsel, Isabelle
- Vincent, Christelle
- Nathan, Nathalie
- SpringerLink (Online service)
Other Objects (12)
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sFlt-1 als diagnostischer Biomarker bei pulmonaler Hypertonie
Regulation of soluble vascular endothelial growth factor receptor (sFlt-1/sVEGFR-1) expression and release in endothelial cells by human follicular fluid and granulosa cells
Regulation of soluble vascular endothelial growth factor receptor (sFlt-1/sVEGFR-1) expression and release in endothelial cells by human follicular fluid and granulosa cells
Damage of the endothelial glycocalyx and cortical stiffening - two new mechanisms of action of soluble Fms-like tyrosine kinase (sFlt-1) contributing to endothelial dysfunction beyond angiogenesis
Autocrine activity of soluble Flt-1 controls endothelial cell function and angiogenesis
Human umbilical cord mesenchymal stem cell derived exosomes (HUCMSC-exos) recovery soluble fms-like tyrosine kinase-1 (sFlt-1)-induced endothelial dysfunction in preeclampsia
Unfractionated heparin displaces sFlt-1 from the placental extracellular matrix
Resveratrol inhibits release of soluble fms-like tyrosine kinase (sFlt-1) and soluble endoglin and improves vascular dysfunction – implications as a preeclampsia treatment
Effects of simvastatin, rosuvastatin and pravastatin on soluble fms-like tyrosine kinase 1 (sFlt-1) and soluble endoglin (sENG) secretion from human umbilical vein endothelial cells, primary trophoblast cells and placenta
Klonierung und regulierbare Expression des löslichen VEGF-Rezeptors sFlt-1 in vitro
sFlt-1 und PIGF als diagnostische und prognostische Marker in der pulmonalen Hypertonie
Alterations in plasma soluble vascular endothelial growth factor receptor-1 (sFlt-1) concentrations during coronary artery bypass graft surgery: relationships with post-operative complications
sFlt-1 als diagnostischer Biomarker bei pulmonaler Hypertonie
Regulation of soluble vascular endothelial growth factor receptor (sFlt-1/sVEGFR-1) expression and release in endothelial cells by human follicular fluid and granulosa cells
Regulation of soluble vascular endothelial growth factor receptor (sFlt-1/sVEGFR-1) expression and release in endothelial cells by human follicular fluid and granulosa cells
Damage of the endothelial glycocalyx and cortical stiffening - two new mechanisms of action of soluble Fms-like tyrosine kinase (sFlt-1) contributing to endothelial dysfunction beyond angiogenesis
Autocrine activity of soluble Flt-1 controls endothelial cell function and angiogenesis
Human umbilical cord mesenchymal stem cell derived exosomes (HUCMSC-exos) recovery soluble fms-like tyrosine kinase-1 (sFlt-1)-induced endothelial dysfunction in preeclampsia
Unfractionated heparin displaces sFlt-1 from the placental extracellular matrix
Resveratrol inhibits release of soluble fms-like tyrosine kinase (sFlt-1) and soluble endoglin and improves vascular dysfunction – implications as a preeclampsia treatment
Effects of simvastatin, rosuvastatin and pravastatin on soluble fms-like tyrosine kinase 1 (sFlt-1) and soluble endoglin (sENG) secretion from human umbilical vein endothelial cells, primary trophoblast cells and placenta
Klonierung und regulierbare Expression des löslichen VEGF-Rezeptors sFlt-1 in vitro
sFlt-1 und PIGF als diagnostische und prognostische Marker in der pulmonalen Hypertonie
Alterations in plasma soluble vascular endothelial growth factor receptor-1 (sFlt-1) concentrations during coronary artery bypass graft surgery: relationships with post-operative complications
sFlt-1 als diagnostischer Biomarker bei pulmonaler Hypertonie
Regulation of soluble vascular endothelial growth factor receptor (sFlt-1/sVEGFR-1) expression and release in endothelial cells by human follicular fluid and granulosa cells
Regulation of soluble vascular endothelial growth factor receptor (sFlt-1/sVEGFR-1) expression and release in endothelial cells by human follicular fluid and granulosa cells
Damage of the endothelial glycocalyx and cortical stiffening - two new mechanisms of action of soluble Fms-like tyrosine kinase (sFlt-1) contributing to endothelial dysfunction beyond angiogenesis
Autocrine activity of soluble Flt-1 controls endothelial cell function and angiogenesis
Human umbilical cord mesenchymal stem cell derived exosomes (HUCMSC-exos) recovery soluble fms-like tyrosine kinase-1 (sFlt-1)-induced endothelial dysfunction in preeclampsia
Unfractionated heparin displaces sFlt-1 from the placental extracellular matrix
Resveratrol inhibits release of soluble fms-like tyrosine kinase (sFlt-1) and soluble endoglin and improves vascular dysfunction – implications as a preeclampsia treatment
Effects of simvastatin, rosuvastatin and pravastatin on soluble fms-like tyrosine kinase 1 (sFlt-1) and soluble endoglin (sENG) secretion from human umbilical vein endothelial cells, primary trophoblast cells and placenta
Klonierung und regulierbare Expression des löslichen VEGF-Rezeptors sFlt-1 in vitro