- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Bibliographic citation
-
Niacin Inhibits Apoptosis and Rescues Premature Ovarian Failure ; volume:50 ; number:6 ; year:2018 ; pages:2060-2070
Cellular physiology and biochemistry ; 50, Heft 6 (2018), 2060-2070
- Creator
-
Wang, Shufang
Sun, Min
Yu, Ling
Wang, Yixuan
Yao, Yuanqing
Wang, Deqing
- DOI
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10.1159/000495051
- URN
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urn:nbn:de:101:1-2018112823110046468492
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:58 AM CEST
Data provider
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Associated
- Wang, Shufang
- Sun, Min
- Yu, Ling
- Wang, Yixuan
- Yao, Yuanqing
- Wang, Deqing
Other Objects (12)
Premature ovarian failure
tsRNA-3043a intensifies apoptosis and senescence of ovarian granulosa cells to drive premature ovarian failure by targeting FLT1
Genetik des Premature-ovarian-failure-Syndroms
Treatment Concepts for Premature Ovarian Failure
Nodal induces apoptosis and inhibits proliferation in ovarian endometriosis-clear cell carcinoma lesions
Cai’s prescription inhibits granulosa cell apoptosis through ARHGAP4 on poor ovarian responders
Garcinone E induces apoptosis and inhibits migration and invasion in ovarian cancer cells
lncRNA-FMR6 directly binds SAV1 to increase apoptosis of granulosa cells in premature ovarian failure
Plasma metabolomic characterization of premature ovarian insufficiency
hUCMSCs reduce theca interstitial cells apoptosis and restore ovarian function in premature ovarian insufficiency rats through regulating NR4A1-mediated mitochondrial mechanisms
TP63 truncating mutation causes increased cell apoptosis and premature ovarian insufficiency by enhanced transcriptional activation of CLCA2
Endometriosis in women undergoing ovarian tissue transplantation due to premature menopause after gonadotoxic treatment or spontaneous premature ovarian failure
Premature ovarian failure
tsRNA-3043a intensifies apoptosis and senescence of ovarian granulosa cells to drive premature ovarian failure by targeting FLT1
Genetik des Premature-ovarian-failure-Syndroms
Treatment Concepts for Premature Ovarian Failure
Nodal induces apoptosis and inhibits proliferation in ovarian endometriosis-clear cell carcinoma lesions
Cai’s prescription inhibits granulosa cell apoptosis through ARHGAP4 on poor ovarian responders
Garcinone E induces apoptosis and inhibits migration and invasion in ovarian cancer cells
lncRNA-FMR6 directly binds SAV1 to increase apoptosis of granulosa cells in premature ovarian failure
Plasma metabolomic characterization of premature ovarian insufficiency
hUCMSCs reduce theca interstitial cells apoptosis and restore ovarian function in premature ovarian insufficiency rats through regulating NR4A1-mediated mitochondrial mechanisms
TP63 truncating mutation causes increased cell apoptosis and premature ovarian insufficiency by enhanced transcriptional activation of CLCA2
Endometriosis in women undergoing ovarian tissue transplantation due to premature menopause after gonadotoxic treatment or spontaneous premature ovarian failure
Premature ovarian failure
tsRNA-3043a intensifies apoptosis and senescence of ovarian granulosa cells to drive premature ovarian failure by targeting FLT1
Genetik des Premature-ovarian-failure-Syndroms
Treatment Concepts for Premature Ovarian Failure
Nodal induces apoptosis and inhibits proliferation in ovarian endometriosis-clear cell carcinoma lesions
Cai’s prescription inhibits granulosa cell apoptosis through ARHGAP4 on poor ovarian responders
Garcinone E induces apoptosis and inhibits migration and invasion in ovarian cancer cells
lncRNA-FMR6 directly binds SAV1 to increase apoptosis of granulosa cells in premature ovarian failure
Plasma metabolomic characterization of premature ovarian insufficiency
hUCMSCs reduce theca interstitial cells apoptosis and restore ovarian function in premature ovarian insufficiency rats through regulating NR4A1-mediated mitochondrial mechanisms
TP63 truncating mutation causes increased cell apoptosis and premature ovarian insufficiency by enhanced transcriptional activation of CLCA2