Antiplatelet effects of aspirin are not affected by the soluble guanylate cyclase activator cinaciguat (BAY 58-2667)
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2210
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
Antiplatelet effects of aspirin are not affected by the soluble guanylate cyclase activator cinaciguat (BAY 58-2667) ; volume:11 ; number:1 ; day:1 ; month:8 ; year:2011 ; pages:1-2 ; date:12.2011
BMC pharmacology ; 11, Heft 1 (1.8.2011), 1-2, 12.2011
- Creator
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Frey, Reiner
Mueck, Wolfgang
Becka, Michael
Kraetzschmar, Joern
Wensing, Georg
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/1471-2210-11-S1-P26
- URN
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urn:nbn:de:101:1-2019062620021085458659
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:58 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Frey, Reiner
- Mueck, Wolfgang
- Becka, Michael
- Kraetzschmar, Joern
- Wensing, Georg
- SpringerLink (Online service)
Other Objects (12)
Structural studies of soluble guanylate cyclase
Binding of [3H]BAY 58-2667 to purified bovine lung soluble guanylate cyclase by scintillation proximity assay
Molecular steps in soluble guanylate cyclase activation
Lessons from soluble guanylate cyclase alpha1 knockouts
The role of soluble guanylate cyclase in metabolism
The soluble guanylate cyclase activator BAY 58-2667 protects against morbidity and mortality in endotoxic shock by recoupling organ systems
Stimulation of soluble guanylate cyclase reduces experimental dermal fibrosis
Role of soluble guanylate cyclase in progressive renal fibrosis
Co-activation of soluble and particulate guanylate cyclase by BAY 58-2667 and BNP enhances cardiorenal function in experimental heart failure
BAY 58–2667, a soluble guanylate cyclase activator, improves cardiopulmonary haemodynamics in acute decompensated heart failure and has a favourable safety profile
BAY 58–2667, a soluble guanylate cyclase activator, has a favourable safety profile and reduces peripheral vascular resistance in healthy male volunteers
Agonist antibody to guanylate cyclase receptor NPR1 regulates vascular tone
Structural studies of soluble guanylate cyclase
Binding of [3H]BAY 58-2667 to purified bovine lung soluble guanylate cyclase by scintillation proximity assay
Molecular steps in soluble guanylate cyclase activation
Lessons from soluble guanylate cyclase alpha1 knockouts
The role of soluble guanylate cyclase in metabolism
The soluble guanylate cyclase activator BAY 58-2667 protects against morbidity and mortality in endotoxic shock by recoupling organ systems
Stimulation of soluble guanylate cyclase reduces experimental dermal fibrosis
Role of soluble guanylate cyclase in progressive renal fibrosis
Co-activation of soluble and particulate guanylate cyclase by BAY 58-2667 and BNP enhances cardiorenal function in experimental heart failure
BAY 58–2667, a soluble guanylate cyclase activator, improves cardiopulmonary haemodynamics in acute decompensated heart failure and has a favourable safety profile
BAY 58–2667, a soluble guanylate cyclase activator, has a favourable safety profile and reduces peripheral vascular resistance in healthy male volunteers
Agonist antibody to guanylate cyclase receptor NPR1 regulates vascular tone
Structural studies of soluble guanylate cyclase
Binding of [3H]BAY 58-2667 to purified bovine lung soluble guanylate cyclase by scintillation proximity assay
Molecular steps in soluble guanylate cyclase activation
Lessons from soluble guanylate cyclase alpha1 knockouts
The role of soluble guanylate cyclase in metabolism
The soluble guanylate cyclase activator BAY 58-2667 protects against morbidity and mortality in endotoxic shock by recoupling organ systems
Stimulation of soluble guanylate cyclase reduces experimental dermal fibrosis
Role of soluble guanylate cyclase in progressive renal fibrosis
Co-activation of soluble and particulate guanylate cyclase by BAY 58-2667 and BNP enhances cardiorenal function in experimental heart failure
BAY 58–2667, a soluble guanylate cyclase activator, improves cardiopulmonary haemodynamics in acute decompensated heart failure and has a favourable safety profile
BAY 58–2667, a soluble guanylate cyclase activator, has a favourable safety profile and reduces peripheral vascular resistance in healthy male volunteers