- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2041-1723
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Structural basis for the second step of group II intron splicing ; volume:9 ; number:1 ; day:8 ; month:11 ; year:2018 ; pages:1-10 ; date:12.2018
Nature Communications ; 9, Heft 1 (8.11.2018), 1-10, 12.2018
- Classification
-
Biowissenschaften, Biologie
- Creator
-
Chan, Russell T.
Peters, Jessica K.
Robart, Aaron R.
Wiryaman, Timothy
Rajashankar, Kanagalaghatta R.
Toor, Navtej
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1038/s41467-018-06678-0
- URN
-
urn:nbn:de:101:1-2018122119563317370230
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:53 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Chan, Russell T.
- Peters, Jessica K.
- Robart, Aaron R.
- Wiryaman, Timothy
- Rajashankar, Kanagalaghatta R.
- Toor, Navtej
- SpringerLink (Online service)
Other Objects (12)
Giant group I intron in a mitochondrial genome is removed by RNA back-splicing
Modern splicing : shown step by step
A Group I Intron Riboswitch
Minor intron splicing revisited: identification of new minor intron-containing genes and tissue-dependent retention and alternative splicing of minor introns
Structural insights into intron catalysis and dynamics during splicing
An axonemal intron splicing program sustains Plasmodium male development
The debranching enzyme Dbr1 regulates lariat turnover and intron splicing
Alternative splicing level related to intron size and organism complexity
An intragenic mutagenesis strategy in Physcomitrella patens to preserve intron splicing
The leader intron of AtMHX can elicit, in the absence of splicing, low-level intron-mediated enhancement that depends on the internal intron sequence
Evaluation of pathogenicity of WT1 intron variants by in vitro splicing analysis
Comprehensive analysis of the Kinetoplastea intron landscape reveals a novel intron-containing gene and the first exclusively trans-splicing eukaryote
Giant group I intron in a mitochondrial genome is removed by RNA back-splicing
Modern splicing : shown step by step
A Group I Intron Riboswitch
Minor intron splicing revisited: identification of new minor intron-containing genes and tissue-dependent retention and alternative splicing of minor introns
Structural insights into intron catalysis and dynamics during splicing
An axonemal intron splicing program sustains Plasmodium male development
The debranching enzyme Dbr1 regulates lariat turnover and intron splicing
Alternative splicing level related to intron size and organism complexity
An intragenic mutagenesis strategy in Physcomitrella patens to preserve intron splicing
The leader intron of AtMHX can elicit, in the absence of splicing, low-level intron-mediated enhancement that depends on the internal intron sequence
Evaluation of pathogenicity of WT1 intron variants by in vitro splicing analysis
Comprehensive analysis of the Kinetoplastea intron landscape reveals a novel intron-containing gene and the first exclusively trans-splicing eukaryote
Giant group I intron in a mitochondrial genome is removed by RNA back-splicing
Modern splicing : shown step by step
A Group I Intron Riboswitch
Minor intron splicing revisited: identification of new minor intron-containing genes and tissue-dependent retention and alternative splicing of minor introns
Structural insights into intron catalysis and dynamics during splicing
An axonemal intron splicing program sustains Plasmodium male development
The debranching enzyme Dbr1 regulates lariat turnover and intron splicing
Alternative splicing level related to intron size and organism complexity
An intragenic mutagenesis strategy in Physcomitrella patens to preserve intron splicing
The leader intron of AtMHX can elicit, in the absence of splicing, low-level intron-mediated enhancement that depends on the internal intron sequence
Evaluation of pathogenicity of WT1 intron variants by in vitro splicing analysis