Irisin promotes the proliferation and tenogenic differentiation of rat tendon-derived stem/progenitor cells via activating YAP/TAZ
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1543-706X
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
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online resource.
- Erschienen in
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Irisin promotes the proliferation and tenogenic differentiation of rat tendon-derived stem/progenitor cells via activating YAP/TAZ ; volume:58 ; number:8 ; day:20 ; month:9 ; year:2022 ; pages:658-668 ; date:9.2022
In vitro cellular & developmental biology / Animal. Animal ; 58, Heft 8 (20.9.2022), 658-668, 9.2022
- Urheber
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Xu, Langhai
Chen, Zhonggai
Geng, Tingting
Ru, Bin
Wan, Quan
Zhang, Jianbin
Li, Shun
Cai, Wenjun
- Beteiligte Personen und Organisationen
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SpringerLink (Online service)
- DOI
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10.1007/s11626-022-00699-2
- URN
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urn:nbn:de:101:1-2022122721044545211615
- Rechteinformation
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:25 MESZ
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Beteiligte
- Xu, Langhai
- Chen, Zhonggai
- Geng, Tingting
- Ru, Bin
- Wan, Quan
- Zhang, Jianbin
- Li, Shun
- Cai, Wenjun
- SpringerLink (Online service)
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YAP/TAZ drives Notch and angiogenesis mechanoregulation in silico
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Hippo-YAP/TAZ signaling in neural crest development and neuroblastoma
Role of YAP/TAZ in endothelial cell growth and metabolism
DNMT3A Cooperates with YAP/TAZ to Drive Gallbladder Cancer Metastasis
AMPK stimulation inhibits YAP/TAZ signaling to ameliorate hepatic fibrosis
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Radial extracorporeal shockwave promotes subchondral bone stem/progenitor cell self-renewal by activating YAP/TAZ and facilitates cartilage repair in vivo
Noncanonical Wnt5a signaling regulates tendon stem/progenitor cells senescence
YAP/TAZ drives Notch and angiogenesis mechanoregulation in silico
PEDF-derived peptide promotes tendon regeneration through its mitogenic effect on tendon stem/progenitor cells
Hippo-YAP/TAZ signaling in neural crest development and neuroblastoma
Role of YAP/TAZ in endothelial cell growth and metabolism
DNMT3A Cooperates with YAP/TAZ to Drive Gallbladder Cancer Metastasis
AMPK stimulation inhibits YAP/TAZ signaling to ameliorate hepatic fibrosis
The role of YAP/TAZ activity in cancer metabolic reprogramming
YAP and TAZ are dispensable for physiological and malignant haematopoiesis
A feed forward loop enforces YAP/TAZ signaling during tumorigenesis