AZD2014, a dual mTOR inhibitor, attenuates cardiac hypertrophy in vitro and in vivo

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1754-1611

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: AZD2014, a dual mTOR inhibitor, attenuates cardiac hypertrophy in vitro and in vivo ; volume:15 ; number:1 ; day:21 ; month:10 ; year:2021 ; pages:1-10 ; date:12.2021
Journal of biological engineering ; 15, Heft 1 (21.10.2021), 1-10, 12.2021

Creator: Cha, Byung-Hyun
Jung, Minjin
Kim, Angela S.
Lepak, Victoria C.
Colson, Brett A.
Bull, David A.
Won, Youngwook

Contributor: SpringerLink (Online service)

DOI: 10.1186/s13036-021-00276-3

URN: urn:nbn:de:101:1-2022011419550073168418

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:39 AM CEST

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Associated

Cha, Byung-Hyun
Jung, Minjin
Kim, Angela S.
Lepak, Victoria C.
Colson, Brett A.
Bull, David A.
Won, Youngwook
SpringerLink (Online service)

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AZD2014, a dual mTOR inhibitor, attenuates cardiac hypertrophy in vitro and in vivo

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Other Objects (12)

Alamandine attenuates hypertension and cardiac hypertrophy in hypertensive rats

E3 ubiquitin ligase RNF5 attenuates pathological cardiac hypertrophy through STING

Antiandrogenic therapy with finasteride attenuates cardiac hypertrophy and left ventricular dysfunction

KLK11 promotes the activation of mTOR and protein synthesis to facilitate cardiac hypertrophy

Roselle attenuates cardiac hypertrophy after myocardial infarction in vivo and in vitro

Arginyltransferase knockdown attenuates cardiac hypertrophy and fibrosis through TAK1-JNK1/2 pathway

Qiliqiangxin Attenuates Phenylephrine-Induced Cardiac Hypertrophy through Downregulation of MiR-199a-5p

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Author Correction: Arginyltransferase knockdown attenuates cardiac hypertrophy and fibrosis through TAK1-JNK1/2 pathway

Circular RNA-circPan3 attenuates cardiac hypertrophy via miR-320-3p/HSP20 axis

I prostanoid receptor activation attenuates pressure overload-induced cardiac hypertrophy by enhancing glucose oxidation

Alamandine attenuates hypertension and cardiac hypertrophy in hypertensive rats

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Antiandrogenic therapy with finasteride attenuates cardiac hypertrophy and left ventricular dysfunction

KLK11 promotes the activation of mTOR and protein synthesis to facilitate cardiac hypertrophy

Roselle attenuates cardiac hypertrophy after myocardial infarction in vivo and in vitro

Arginyltransferase knockdown attenuates cardiac hypertrophy and fibrosis through TAK1-JNK1/2 pathway

Qiliqiangxin Attenuates Phenylephrine-Induced Cardiac Hypertrophy through Downregulation of MiR-199a-5p

Cardiac hypertrophy at autopsy

The orphan receptor TR3 participates in angiotensin II‐induced cardiac hypertrophy by controlling mTOR signalling

Author Correction: Arginyltransferase knockdown attenuates cardiac hypertrophy and fibrosis through TAK1-JNK1/2 pathway

Circular RNA-circPan3 attenuates cardiac hypertrophy via miR-320-3p/HSP20 axis

I prostanoid receptor activation attenuates pressure overload-induced cardiac hypertrophy by enhancing glucose oxidation

Alamandine attenuates hypertension and cardiac hypertrophy in hypertensive rats

E3 ubiquitin ligase RNF5 attenuates pathological cardiac hypertrophy through STING

Antiandrogenic therapy with finasteride attenuates cardiac hypertrophy and left ventricular dysfunction

KLK11 promotes the activation of mTOR and protein synthesis to facilitate cardiac hypertrophy

Roselle attenuates cardiac hypertrophy after myocardial infarction in vivo and in vitro

Arginyltransferase knockdown attenuates cardiac hypertrophy and fibrosis through TAK1-JNK1/2 pathway

Qiliqiangxin Attenuates Phenylephrine-Induced Cardiac Hypertrophy through Downregulation of MiR-199a-5p

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The orphan receptor TR3 participates in angiotensin II‐induced cardiac hypertrophy by controlling mTOR signalling

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Circular RNA-circPan3 attenuates cardiac hypertrophy via miR-320-3p/HSP20 axis

I prostanoid receptor activation attenuates pressure overload-induced cardiac hypertrophy by enhancing glucose oxidation

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