Long-term PGC1β overexpression leads to apoptosis, autophagy and muscle wasting

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2045-2322

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Long-term PGC1β overexpression leads to apoptosis, autophagy and muscle wasting ; volume:7 ; number:1 ; day:31 ; month:8 ; year:2017 ; pages:1-16 ; date:12.2017
Scientific reports ; 7, Heft 1 (31.8.2017), 1-16, 12.2017

Creator: Sopariwala, Danesh H.
Yadav, Vikas
Badin, Pierre-Marie
Likhite, Neah
Sheth, Megha
Lorca, Sabina
Vila, Isabelle K.
Kim, Eun Ran
Tong, Qingchun
Song, Min Sup
Rodney, George G.
Narkar, Vihang A.

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41598-017-10238-9

URN: urn:nbn:de:101:1-2019102016092919792707

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:36 AM CEST

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Associated

Sopariwala, Danesh H.
Yadav, Vikas
Badin, Pierre-Marie
Likhite, Neah
Sheth, Megha
Lorca, Sabina
Vila, Isabelle K.
Kim, Eun Ran
Tong, Qingchun
Song, Min Sup
Rodney, George G.
Narkar, Vihang A.
SpringerLink (Online service)

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Long-term PGC1β overexpression leads to apoptosis, autophagy and muscle wasting

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Other Objects (12)

BAG3 overexpression and cytoprotective autophagy mediate apoptosis resistance in chemoresistant breast cancer cells

Niclosamide ethanolamine ameliorates diabetes-related muscle wasting by inhibiting autophagy

Increased autophagy leads to decreased apoptosis during β-thalassaemic mouse and patient erythropoiesis

Host autophagy mediates organ wasting and nutrient mobilization for tumor growth

Overexpression of Programmed Cell Death 1 Prevents Doxorubicin-Induced Apoptosis Through Autophagy Induction in H9c2 Cardiomyocytes

Pim1 Overexpression Prevents Apoptosis in Cardiomyocytes After Exposure to Hypoxia and Oxidative Stress via Upregulating Cell Autophagy

Apoptosis, autophagy, necroptosis, and cancer metastasis

Overexpression of CPEB3 leads to astrocyte dysfunction

Overexpression of CPEB3 leads to astrocyte dysfunction

BRCA mutations lead to XIAP overexpression and sensitise ovarian cancer to inhibitor of apoptosis (IAP) family inhibitors

Overexpression of IκBα in cardiomyocytes alleviates hydrogen peroxide-induced apoptosis and autophagy by inhibiting NF-κB activation

Heat shock protein A4 ablation leads to skeletal muscle myopathy associated with dysregulated autophagy and induced apoptosis

BAG3 overexpression and cytoprotective autophagy mediate apoptosis resistance in chemoresistant breast cancer cells

Niclosamide ethanolamine ameliorates diabetes-related muscle wasting by inhibiting autophagy

Increased autophagy leads to decreased apoptosis during β-thalassaemic mouse and patient erythropoiesis

Host autophagy mediates organ wasting and nutrient mobilization for tumor growth

Overexpression of Programmed Cell Death 1 Prevents Doxorubicin-Induced Apoptosis Through Autophagy Induction in H9c2 Cardiomyocytes

Pim1 Overexpression Prevents Apoptosis in Cardiomyocytes After Exposure to Hypoxia and Oxidative Stress via Upregulating Cell Autophagy

Apoptosis, autophagy, necroptosis, and cancer metastasis

Overexpression of CPEB3 leads to astrocyte dysfunction

Overexpression of CPEB3 leads to astrocyte dysfunction

BRCA mutations lead to XIAP overexpression and sensitise ovarian cancer to inhibitor of apoptosis (IAP) family inhibitors

Overexpression of IκBα in cardiomyocytes alleviates hydrogen peroxide-induced apoptosis and autophagy by inhibiting NF-κB activation

Heat shock protein A4 ablation leads to skeletal muscle myopathy associated with dysregulated autophagy and induced apoptosis

BAG3 overexpression and cytoprotective autophagy mediate apoptosis resistance in chemoresistant breast cancer cells

Niclosamide ethanolamine ameliorates diabetes-related muscle wasting by inhibiting autophagy

Increased autophagy leads to decreased apoptosis during β-thalassaemic mouse and patient erythropoiesis

Host autophagy mediates organ wasting and nutrient mobilization for tumor growth

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Pim1 Overexpression Prevents Apoptosis in Cardiomyocytes After Exposure to Hypoxia and Oxidative Stress via Upregulating Cell Autophagy

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Overexpression of IκBα in cardiomyocytes alleviates hydrogen peroxide-induced apoptosis and autophagy by inhibiting NF-κB activation

Heat shock protein A4 ablation leads to skeletal muscle myopathy associated with dysregulated autophagy and induced apoptosis

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