HSP60 critically regulates endogenous IL-1β production in activated microglia by stimulating NLRP3 inflammasome pathway
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1742-2094
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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HSP60 critically regulates endogenous IL-1β production in activated microglia by stimulating NLRP3 inflammasome pathway ; volume:15 ; number:1 ; day:9 ; month:6 ; year:2018 ; pages:1-19 ; date:12.2018
Journal of neuroinflammation ; 15, Heft 1 (9.6.2018), 1-19, 12.2018
- Creator
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Swaroop, Shalini
- Contributor
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Mahadevan, Anita
Shankar, Susarla Krishna
Adlakha, Yogita K.
Basu, Anirban
SpringerLink (Online service)
- DOI
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10.1186/s12974-018-1214-5
- URN
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urn:nbn:de:101:1-2018081604250962627987
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
- 15.08.2025, 7:35 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Swaroop, Shalini
- Mahadevan, Anita
- Shankar, Susarla Krishna
- Adlakha, Yogita K.
- Basu, Anirban
- SpringerLink (Online service)
Other Objects (12)
Correction to: HSP60 critically regulates endogenous IL-1β production in activated microglia by stimulating NLRP3 inflammasome pathway
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STAT3 promotes NLRP3 inflammasome activation by mediating NLRP3 mitochondrial translocation
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Role of the NLRP3 inflammasome in cancer
Correction to: HSP60 critically regulates endogenous IL-1β production in activated microglia by stimulating NLRP3 inflammasome pathway
A20 critically controls microglia activation and inhibits inflammasome-dependent neuroinflammation
A20 critically controls microglia activation and inhibits inflammasome-dependent neuroinflammation
Procyanidins alleviates morphine tolerance by inhibiting activation of NLRP3 inflammasome in microglia
Tenuigenin protects dopaminergic neurons from inflammation via suppressing NLRP3 inflammasome activation in microglia
Tofacitinib inhibits granulocyte–macrophage colony-stimulating factor-induced NLRP3 inflammasome activation in human neutrophils
SARS-CoV-2 drives NLRP3 inflammasome activation in human microglia through spike protein
MPTP-driven NLRP3 inflammasome activation in microglia plays a central role in dopaminergic neurodegeneration
Corylin inhibits LPS-induced inflammatory response and attenuates the activation of NLRP3 inflammasome in microglia
STAT3 promotes NLRP3 inflammasome activation by mediating NLRP3 mitochondrial translocation
UAF1 deubiquitinase complexes facilitate NLRP3 inflammasome activation by promoting NLRP3 expression
Role of the NLRP3 inflammasome in cancer
Correction to: HSP60 critically regulates endogenous IL-1β production in activated microglia by stimulating NLRP3 inflammasome pathway
A20 critically controls microglia activation and inhibits inflammasome-dependent neuroinflammation
A20 critically controls microglia activation and inhibits inflammasome-dependent neuroinflammation
Procyanidins alleviates morphine tolerance by inhibiting activation of NLRP3 inflammasome in microglia
Tenuigenin protects dopaminergic neurons from inflammation via suppressing NLRP3 inflammasome activation in microglia
Tofacitinib inhibits granulocyte–macrophage colony-stimulating factor-induced NLRP3 inflammasome activation in human neutrophils
SARS-CoV-2 drives NLRP3 inflammasome activation in human microglia through spike protein
MPTP-driven NLRP3 inflammasome activation in microglia plays a central role in dopaminergic neurodegeneration
Corylin inhibits LPS-induced inflammatory response and attenuates the activation of NLRP3 inflammasome in microglia
STAT3 promotes NLRP3 inflammasome activation by mediating NLRP3 mitochondrial translocation
UAF1 deubiquitinase complexes facilitate NLRP3 inflammasome activation by promoting NLRP3 expression