Biphasic onset of splenic apoptosis following hemorrhagic shock: critical implications for Bax, Bcl-2, and Mcl-1 proteins
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1364-8535
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
Biphasic onset of splenic apoptosis following hemorrhagic shock: critical implications for Bax, Bcl-2, and Mcl-1 proteins ; volume:12 ; number:1 ; day:22 ; month:1 ; year:2008 ; pages:1-10 ; date:2.2008
Critical care ; 12, Heft 1 (22.1.2008), 1-10, 2.2008
- Creator
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Hostmann, Arwed
- Contributor
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Jasse, Kerstin
Schulze-Tanzil, Gundula
Robinson, Yohan
Oberholzer, Andreas
Ertel, Wolfgang
Tschoeke, Sven K.
SpringerLink (Online service)
- DOI
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10.1186/cc6772
- URN
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urn:nbn:de:1111-2016080818899
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
- 14.08.2025, 10:45 AM CEST
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Associated
- Hostmann, Arwed
- Jasse, Kerstin
- Schulze-Tanzil, Gundula
- Robinson, Yohan
- Oberholzer, Andreas
- Ertel, Wolfgang
- Tschoeke, Sven K.
- SpringerLink (Online service)
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BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival
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Glutathione and Bcl-2 targeting facilitates elimination by chemoradiotherapy of human A375 melanoma xenografts overexpressing bcl-xl, bcl-2, and mcl-1
Das Bak/Bax homologe Bcl-2 Protein Bok : ein maßgeblicher Apoptoseregulator
Korrelation bekannter Prognosefaktoren des Mammakarzonoms mit p53, bcl-2 und bax
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Epigenetic targeting of Mcl-1 Is synthetically lethal with Bcl-xL/Bcl-2 inhibition in model systems of glioblastoma
MCL.
Boron neutron capture therapy induces apoptosis of glioma cells through Bcl-2/Bax
A direct comparison of selective BH3-mimetics reveals BCL-XL, BCL-2 and MCL-1 as promising therapeutic targets in neuroblastoma