NPM-hMLF1 fusion protein suppresses defects of a Drosophila FTLD model expressing the human FUS gene
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2045-2322
- Extent
-
Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
NPM-hMLF1 fusion protein suppresses defects of a Drosophila FTLD model expressing the human FUS gene ; volume:8 ; number:1 ; day:26 ; month:7 ; year:2018 ; pages:1-14 ; date:12.2018
Scientific reports ; 8, Heft 1 (26.7.2018), 1-14, 12.2018
- Classification
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Biowissenschaften, Biologie
- Creator
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Yamamoto, Itaru
- Contributor
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Azuma, Yumiko
Kushimura, Yukie
Yoshida, Hideki
Mizuta, Ikuko
Mizuno, Toshiki
Ueyama, Morio
Nagai, Yoshitaka
Tokuda, Takahiko
Yamaguchi, Masamitsu
SpringerLink (Online service)
- DOI
-
10.1038/s41598-018-29716-9
- URN
-
urn:nbn:de:101:1-2018092322380285713765
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
- 15.08.2025, 7:22 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Yamamoto, Itaru
- Azuma, Yumiko
- Kushimura, Yukie
- Yoshida, Hideki
- Mizuta, Ikuko
- Mizuno, Toshiki
- Ueyama, Morio
- Nagai, Yoshitaka
- Tokuda, Takahiko
- Yamaguchi, Masamitsu
- SpringerLink (Online service)
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Enhancing folic acid metabolism suppresses defects associated with loss of Drosophila mitofusin
Transcription through enhancers suppresses their activity in Drosophila
Dynamic Polycomb Chromatin Suppresses Aberrant Transcription in Drosophila Immunity
Escargot maintains stemness and suppresses differentiation in Drosophila intestinal stem cells
Activation of arcuate nucleus glucagon-like peptide-1 receptor-expressing neurons suppresses food intake
FUS-linked essential tremor associated with motor dysfunction in Drosophila
The autophagy protein Def8 is altered in Alzheimer's disease and Aβ42-expressing Drosophila brains
A gut-derived hormone suppresses sugar appetite and regulates food choice in Drosophila
Reduced oxidative stress suppresses neurotoxicity in the Drosophila model of TAF15-associated proteinopathies
Expression of Ciona intestinalis AOX causes male reproductive defects in Drosophila melanogaster
A small cohort of FRUM and Engrailed-expressing neurons mediate successful copulation in Drosophila melanogaster
Mushroom body miscellanea : transgenic Drosophila strains expressing anatomical and physiological sensor proteins in Kenyon cells
Enhancing folic acid metabolism suppresses defects associated with loss of Drosophila mitofusin
Transcription through enhancers suppresses their activity in Drosophila
Dynamic Polycomb Chromatin Suppresses Aberrant Transcription in Drosophila Immunity
Escargot maintains stemness and suppresses differentiation in Drosophila intestinal stem cells
Activation of arcuate nucleus glucagon-like peptide-1 receptor-expressing neurons suppresses food intake
FUS-linked essential tremor associated with motor dysfunction in Drosophila
The autophagy protein Def8 is altered in Alzheimer's disease and Aβ42-expressing Drosophila brains
A gut-derived hormone suppresses sugar appetite and regulates food choice in Drosophila
Reduced oxidative stress suppresses neurotoxicity in the Drosophila model of TAF15-associated proteinopathies
Expression of Ciona intestinalis AOX causes male reproductive defects in Drosophila melanogaster
A small cohort of FRUM and Engrailed-expressing neurons mediate successful copulation in Drosophila melanogaster