- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1471-2202
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
parkin counteracts symptoms in a Drosophila model of Parkinson's disease ; volume:5 ; number:1 ; day:16 ; month:4 ; year:2004 ; pages:1-12 ; date:12.2004
BMC neuroscience ; 5, Heft 1 (16.4.2004), 1-12, 12.2004
- Creator
-
Haywood, Annika FM
- Contributor
-
Staveley, Brian E.
SpringerLink (Online service)
- DOI
-
10.1186/1471-2202-5-14
- URN
-
urn:nbn:de:1111-2016032050628
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:45 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Haywood, Annika FM
- Staveley, Brian E.
- SpringerLink (Online service)
Other Objects (12)
Effects of MUL1 and PARKIN on the circadian clock, brain and behaviour in Drosophila Parkinson’s disease models
Mitochondriale Dysfunktion bei Parkin-Mausmodellen des Morbus Parkinson
Deconstructing the molecular genetics behind the PINK1/Parkin axis in Parkinson’s disease using Drosophila melanogaster as a model organism
Quantitative proteomic analysis of Parkin substrates in Drosophila neurons
Multitasking guardian of mitochondrial quality: Parkin function and Parkinson’s disease
Parkinson’s disease-associated protein Parkin: an unusual player in cancer
Parkinson's disease: PINK1, PARKIN and stress signaling to mitochondria
Parkinson’s disease-linked Parkin mutations impair glutamatergic signaling in hippocampal neurons
Short-term temporal memory in idiopathic and Parkin-associated Parkinson’s disease
Zinc supplement greatly improves the condition of parkin mutant Drosophila
Augenbewegungsstörungen bei Parkinson-Syndromen mit und ohne Mutation im Parkin-Gen
Parkin drives pS65‐Ub turnover independently of canonical autophagy in Drosophila
Effects of MUL1 and PARKIN on the circadian clock, brain and behaviour in Drosophila Parkinson’s disease models
Mitochondriale Dysfunktion bei Parkin-Mausmodellen des Morbus Parkinson
Deconstructing the molecular genetics behind the PINK1/Parkin axis in Parkinson’s disease using Drosophila melanogaster as a model organism
Quantitative proteomic analysis of Parkin substrates in Drosophila neurons
Multitasking guardian of mitochondrial quality: Parkin function and Parkinson’s disease
Parkinson’s disease-associated protein Parkin: an unusual player in cancer
Parkinson's disease: PINK1, PARKIN and stress signaling to mitochondria
Parkinson’s disease-linked Parkin mutations impair glutamatergic signaling in hippocampal neurons
Short-term temporal memory in idiopathic and Parkin-associated Parkinson’s disease
Zinc supplement greatly improves the condition of parkin mutant Drosophila
Augenbewegungsstörungen bei Parkinson-Syndromen mit und ohne Mutation im Parkin-Gen
Parkin drives pS65‐Ub turnover independently of canonical autophagy in Drosophila
Effects of MUL1 and PARKIN on the circadian clock, brain and behaviour in Drosophila Parkinson’s disease models
Mitochondriale Dysfunktion bei Parkin-Mausmodellen des Morbus Parkinson
Deconstructing the molecular genetics behind the PINK1/Parkin axis in Parkinson’s disease using Drosophila melanogaster as a model organism
Quantitative proteomic analysis of Parkin substrates in Drosophila neurons
Multitasking guardian of mitochondrial quality: Parkin function and Parkinson’s disease
Parkinson’s disease-associated protein Parkin: an unusual player in cancer
Parkinson's disease: PINK1, PARKIN and stress signaling to mitochondria
Parkinson’s disease-linked Parkin mutations impair glutamatergic signaling in hippocampal neurons
Short-term temporal memory in idiopathic and Parkin-associated Parkinson’s disease
Zinc supplement greatly improves the condition of parkin mutant Drosophila
Augenbewegungsstörungen bei Parkinson-Syndromen mit und ohne Mutation im Parkin-Gen