Collagen VI sustains cell stemness and chemotherapy resistance in glioblastoma
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
Online-Ressource, 1 online resource.
- Sprache
-
Englisch
- Erschienen in
-
Collagen VI sustains cell stemness and chemotherapy resistance in glioblastoma ; volume:80 ; number:8 ; day:28 ; month:7 ; year:2023 ; pages:1-16 ; date:8.2023
Cellular and molecular life sciences ; 80, Heft 8 (28.7.2023), 1-16, 8.2023
- Urheber
-
Cescon, Matilde
Rampazzo, Elena
Bresolin, Silvia
Da Ros, Francesco
Manfreda, Lorenzo
Cani, Alice
Della Puppa, Alessandro
Braghetta, Paola
Bonaldo, Paolo
Persano, Luca
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1007/s00018-023-04887-5
- URN
-
urn:nbn:de:101:1-2023110309553608608783
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
- 14.08.2025, 10:54 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Cescon, Matilde
- Rampazzo, Elena
- Bresolin, Silvia
- Da Ros, Francesco
- Manfreda, Lorenzo
- Cani, Alice
- Della Puppa, Alessandro
- Braghetta, Paola
- Bonaldo, Paolo
- Persano, Luca
- SpringerLink (Online service)
Ähnliche Objekte (12)
EMP3 sustains oncogenic EGFR/CDK2 signaling by restricting receptor degradation in glioblastoma
Suppressing Dazl modulates tumorigenicity and stemness in human glioblastoma cells
Farnesyl diphosphate synthase is important for the maintenance of glioblastoma stemness
CD146 increases stemness and aggressiveness in glioblastoma and activates YAP signaling
IL20RB signaling enhances stemness and chemotherapy resistance in pancreatic cancer
Downregulated CLIP3 induces radioresistance by enhancing stemness and glycolytic flux in glioblastoma
Aporphine and isoquinoline derivatives block glioblastoma cell stemness and enhance temozolomide cytotoxicity
R-spodin2 enhances canonical Wnt signaling to maintain the stemness of glioblastoma cells
Motility of glioblastoma cells is driven by netrin-1 induced gain of stemness
Napabucasin, a novel STAT3 inhibitor suppresses proliferation, invasion and stemness of glioblastoma cells
NME6: ribonucleotide salvage sustains mitochondrial transcription
Different states of stemness of glioblastoma stem cells sustain glioblastoma subtypes indicating novel clinical biomarkers and high-efficacy customized therapies
EMP3 sustains oncogenic EGFR/CDK2 signaling by restricting receptor degradation in glioblastoma
Suppressing Dazl modulates tumorigenicity and stemness in human glioblastoma cells
Farnesyl diphosphate synthase is important for the maintenance of glioblastoma stemness
CD146 increases stemness and aggressiveness in glioblastoma and activates YAP signaling
IL20RB signaling enhances stemness and chemotherapy resistance in pancreatic cancer
Downregulated CLIP3 induces radioresistance by enhancing stemness and glycolytic flux in glioblastoma
Aporphine and isoquinoline derivatives block glioblastoma cell stemness and enhance temozolomide cytotoxicity
R-spodin2 enhances canonical Wnt signaling to maintain the stemness of glioblastoma cells
Motility of glioblastoma cells is driven by netrin-1 induced gain of stemness
Napabucasin, a novel STAT3 inhibitor suppresses proliferation, invasion and stemness of glioblastoma cells
NME6: ribonucleotide salvage sustains mitochondrial transcription
Different states of stemness of glioblastoma stem cells sustain glioblastoma subtypes indicating novel clinical biomarkers and high-efficacy customized therapies
EMP3 sustains oncogenic EGFR/CDK2 signaling by restricting receptor degradation in glioblastoma
Suppressing Dazl modulates tumorigenicity and stemness in human glioblastoma cells
Farnesyl diphosphate synthase is important for the maintenance of glioblastoma stemness
CD146 increases stemness and aggressiveness in glioblastoma and activates YAP signaling
IL20RB signaling enhances stemness and chemotherapy resistance in pancreatic cancer
Downregulated CLIP3 induces radioresistance by enhancing stemness and glycolytic flux in glioblastoma
Aporphine and isoquinoline derivatives block glioblastoma cell stemness and enhance temozolomide cytotoxicity
R-spodin2 enhances canonical Wnt signaling to maintain the stemness of glioblastoma cells
Motility of glioblastoma cells is driven by netrin-1 induced gain of stemness
Napabucasin, a novel STAT3 inhibitor suppresses proliferation, invasion and stemness of glioblastoma cells
NME6: ribonucleotide salvage sustains mitochondrial transcription