Author Correction: Mitochondria-specific drug release and reactive oxygen species burst induced by polyprodrug nanoreactors can enhance chemotherapy
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2041-1723
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Author Correction: Mitochondria-specific drug release and reactive oxygen species burst induced by polyprodrug nanoreactors can enhance chemotherapy ; volume:10 ; number:1 ; day:10 ; month:6 ; year:2019 ; pages:1-2 ; date:12.2019
Nature Communications ; 10, Heft 1 (10.6.2019), 1-2, 12.2019
- Creator
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Zhang, Wenjia
Hu, Xianglong
Shen, Qi
Xing, Da
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41467-019-10186-0
- URN
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urn:nbn:de:101:1-2019102122213409362245
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
- 15.08.2025, 7:37 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Zhang, Wenjia
- Hu, Xianglong
- Shen, Qi
- Xing, Da
- SpringerLink (Online service)
Other Objects (12)
Mitochondria-specific drug release and reactive oxygen species burst induced by polyprodrug nanoreactors can enhance chemotherapy
Intracellular Amplifiers of Reactive Oxygen Species Affecting Mitochondria as Radiosensitizers
Extracellular release in the quality control of the mammalian mitochondria
Lactobacilli enhance reactive oxygen species-dependent apoptosis-inducing signaling
Oxygen tank for synergistic hypoxia relief to enhance mitochondria-targeted photodynamic therapy
Translation termination in human mitochondria – substrate specificity of mitochondrial release factors
The role of reactive oxygen species and mitochondria in T-cell activation
Incorporating a Polyethyleneglycol Linker to Enhance the Hydrophilicity of Mitochondria‐Targeted Triphenylphosphonium Constructs
AIP limits neurotransmitter release by inhibiting calcium bursts from the ryanodine receptor
Modulated-diameter zirconia nanotubes for controlled drug release - bye to the burst
Targeted production of reactive oxygen species in mitochondria to overcome cancer drug resistance
Reactive oxygen species stress increases accumulation of tyrosyl-DNA phsosphodiesterase 1 within mitochondria
Mitochondria-specific drug release and reactive oxygen species burst induced by polyprodrug nanoreactors can enhance chemotherapy
Intracellular Amplifiers of Reactive Oxygen Species Affecting Mitochondria as Radiosensitizers
Extracellular release in the quality control of the mammalian mitochondria
Lactobacilli enhance reactive oxygen species-dependent apoptosis-inducing signaling
Oxygen tank for synergistic hypoxia relief to enhance mitochondria-targeted photodynamic therapy
Translation termination in human mitochondria – substrate specificity of mitochondrial release factors
The role of reactive oxygen species and mitochondria in T-cell activation
Incorporating a Polyethyleneglycol Linker to Enhance the Hydrophilicity of Mitochondria‐Targeted Triphenylphosphonium Constructs
AIP limits neurotransmitter release by inhibiting calcium bursts from the ryanodine receptor
Modulated-diameter zirconia nanotubes for controlled drug release - bye to the burst
Targeted production of reactive oxygen species in mitochondria to overcome cancer drug resistance
Reactive oxygen species stress increases accumulation of tyrosyl-DNA phsosphodiesterase 1 within mitochondria
Mitochondria-specific drug release and reactive oxygen species burst induced by polyprodrug nanoreactors can enhance chemotherapy
Intracellular Amplifiers of Reactive Oxygen Species Affecting Mitochondria as Radiosensitizers
Extracellular release in the quality control of the mammalian mitochondria
Lactobacilli enhance reactive oxygen species-dependent apoptosis-inducing signaling
Oxygen tank for synergistic hypoxia relief to enhance mitochondria-targeted photodynamic therapy
Translation termination in human mitochondria – substrate specificity of mitochondrial release factors
The role of reactive oxygen species and mitochondria in T-cell activation
Incorporating a Polyethyleneglycol Linker to Enhance the Hydrophilicity of Mitochondria‐Targeted Triphenylphosphonium Constructs
AIP limits neurotransmitter release by inhibiting calcium bursts from the ryanodine receptor
Modulated-diameter zirconia nanotubes for controlled drug release - bye to the burst
Targeted production of reactive oxygen species in mitochondria to overcome cancer drug resistance